Dr. Michael Gebhard Schlossmacher MD
Neurologist | Neurology
75 Francis Street Partners Parkinson A Boston MA, 02115About
Dr. Michael Schlossmacher is a distinguished Neurologist in Boston, MA. Dr. Schlossmacher specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Schlossmacher employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Schlossmacher is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
Universitat Wien
MEDICAL FACULTY UNIV OF VIENNA 1987
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Seizures after epidural blood patch.
- Dimerization of Parkinson's disease-causing DJ-1 and formation of high molecular weight complexes in human brain.
- The glucocerebrosidase gene and Parkinson's disease in Ashkenazi Jews.
- Parkinson's disease: assays for the ubiquitin ligase activity of neural Parkin.
- Lewy body Parkinson's disease in a large pedigree with 77 Parkin mutation carriers.
- Detection of oligomeric forms of alpha-synuclein protein in human plasma as a potential biomarker for Parkinson's disease.
- Parkin protects against mitochondrial toxins and beta-amyloid accumulation in skeletal muscle cells.
- Phosphorylation of Ser-129 is the dominant pathological modification of alpha-synuclein in familial and sporadic Lewy body disease.
- Decreased alpha-synuclein in cerebrospinal fluid of aged individuals and subjects with Parkinson's disease.
- The genetics of Parkinson disease: Implications for neurological care.
- Structure of acid beta-glucosidase with pharmacological chaperone provides insight into Gaucher disease.
- Serum heart-type fatty acid-binding protein and cerebrospinal fluid tau: marker candidates for dementia with Lewy bodies.
- Parkinson disease, 10 years after its genetic revolution: multiple clues to a complex disorder.
- The effects of oxidative stress on parkin and other E3 ligases.
- Direct quantification of CSF alpha-synuclein by ELISA and first cross-sectional study in patients with neurodegeneration.
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