Dr. Scott C. Kogan M.D.
Pathologist | Anatomic Pathology & Clinical Pathology
505 Parnassus Ave San Francisco CA, 94143About
Dr. Scott Kogan is a pathologist practicing in San Francisco, CA. Dr. Kogan is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Kogan can help your primary care doctor make a diagnosis about your medical condition. Dr. Kogan may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Education and Training
Univ of Ca, San Francisco, Sch of Med, San Francisco Ca 1991
University of California 1991
Board Certification
PathologyAmerican Board of PathologyABP
Internal MedicineAmerican Board of Internal MedicineABIM- Hematology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Retinoic acid and arsenic synergize to eradicate leukemic cells in a mouse model of acute promyelocytic leukemia.
- Images in clinical medicine. Indinavir crystalluria.
- Acute promyelocytic leukemia: from treatment to genetics and back.
- Mouse intestinal goblet cells expressing SV40 T antigen directed by the MUC2 mucin gene promoter undergo apoptosis upon migration to the villi.
- Acute promyelocytic leukemia: a view from a mouse.
- Recurring chromosomal abnormalities in leukemia in PML-RARA transgenic mice parallel human acute promyelocytic leukemia.
- CCAAT/Enhancer binding proteins repress the leukemic phenotype of acute myeloid leukemia.
- A model of APL with FLT3 mutation is responsive to retinoic acid and a receptor tyrosine kinase inhibitor, SU11657.
- Recurring chromosomal abnormalities in leukemia in PML-RARA transgenic mice identify cooperating events and genetic pathways to acute promyelocytic leukemia.
- Cooperation of cytokine signaling with chimeric transcription factors in
- Somatic activation of oncogenic Kras in hematopoietic cells initiates a rapidly fatal myeloproliferative disorder.
- Somatic inactivation of Nf1 in hematopoietic cells results in a progressive myeloproliferative disorder.
- Role of O6-alkylguanine-DNA alkyltransferase in protecting against 1,3-bis(2-chloroethyl)-1-nitrosourea (BCNU)-induced long-term toxicities.
- Induction of tumor arrest and differentiation with prolonged survival by intermittent hypoxia in a mouse model of acute myeloid leukemia.
- Therapy-induced malignant neoplasms in Nf1 mutant mice.
Treatments
- Blood Disorders
Dr. Scott C. Kogan M.D.'s Practice location
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