Dr. Catherine E Keegan MD PHD
Pediatrician
1500 East Medical Center Dr 1st Floor Taubman Ct Ann Arbor MI, 48109About
Dr. Catherine Keegan is a pediatrician practicing in Ann Arbor, MI. Dr. Keegan is a doctor who specializes in the health care of children. As a pedicatrician, Dr. Keegan diagnoses and treats infections, injuries, diseases and other disorders in children. Pediatricians typically work with infants, children, teenagers and young adults up to age 21. They practice medical care as well as preventative health care. Dr. Keegan can oversee and manage the physical, mental and emotional health of their patients.
Education and Training
University of Mi Med Sch 1995
Board Certification
Medical GeneticsAmerican Board of Medical GeneticsABMG
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mouse knockout solves endocrine puzzle and promotes new pituitary lineage model.
- The value of intravenous procaine amide in the treatment of tachycardias.
- IMAGe association and congenital adrenal hypoplasia: no disease-causing mutations found in the ACD gene.
- Craniofacial dyssynostosis in two boys with apparently normal cognitive development.
- Microduplications of 22q11.2 are frequently inherited and are associated with variable phenotypes.
- Identification of critical regions for clinical features of distal 10q deletion syndrome.
- Caudal regression in adrenocortical dysplasia (acd) mice is caused by telomere dysfunction with subsequent p53-dependent apoptosis.
- Telomere protection by TPP1 is mediated by POT1a and POT1b.
- Comprehensive genetic analysis of OEIS complex reveals no evidence for a recurrent microdeletion or duplication.
- Additive effect of TAp63 deficiency on the adrenocortical dysplasia (acd) phenotype.
- Next-generation sequencing identifies the Danforth's short tail mouse mutation as a retrotransposon insertion affecting Ptf1a expression.
- 9p partial monosomy and disorders of sex development: review and postulation of a pathogenetic mechanism.
- Hematopoietic stem cells are acutely sensitive to Acd shelterin gene inactivation.
- Isodicentric Y mosaicism involving a 46, XX cell line: Implications for management.
- De novo dominant ASXL3 mutations alter H2A deubiquitination and transcription in Bainbridge-Ropers syndrome.
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