Dr. Tamala J Bos MD
Psychiatrist | Psychiatry
3355 Eagle Park Dr Ne Grand Rapids MI, 49525About
Dr. Tamala Bos is a psychiatrist practicing in Grand Rapids, MI. Dr. Bos is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. Bos diagnoses and treats mental illnesses. Dr. Bos may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. Bos treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN- Child and Adolescent Psychiatry
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Enhanced cell motility and invasion of chicken embryo fibroblasts in response to Jun over-expression.
- The chicken c-Jun 5' untranslated region directs translation by internal initiation.
- Oncogenes and cell growth.
- Cycloheximide stimulation of cyanide-resistant respiration in suspension cultures
- jun: oncogene and transcription factor.
- Biological and immunological properties of haemagglutinin and neuraminidase expressed from cloned cDNAs in prokaryotic and eukaryotic cells.
- The oncogene jun and nuclear signalling.
- Surface expression of influenza virus neuraminidase, an amino-terminally anchored viral membrane glycoprotein, in polarized epithelial cells.
- Homology between the DNA-binding domain of the GCN4 regulatory protein of yeast and the carboxyl-terminal region of a protein coded for by the oncogene jun.
- Identification of defects in the neuraminidase gene of four temperature-sensitive mutants of A/WSN/33 influenza virus.
- Active influenza virus neuraminidase is expressed in monkey cells from cDNA cloned in simian virus 40 vectors.
- NH2-terminal hydrophobic region of influenza virus neuraminidase provides the signal function in translocation.
- Modifications to the differential display technique reduce background and increase sensitivity.
- In vivo viral and cellular Jun complexes exhibit differential interaction with a number of in vitro generated 'AP-1- and CREB-like' target sequences.
- Isolation and cloning of JTAP-1: a cathepsin like gene upregulated in response to V-Jun induced cell transformation.
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