Dr. Grant D. Barish M.D.
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
3350 La Jolla Village Dr San Diego CA, 92161About
Dr. Grant Barish practices Endocrinology in San Diego, CA. Dr. Barish specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Barish examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ of Mi Med Sch, Ann Arbor Mi 2000
University of Mi Med Sch 1998
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology, Diabetes and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Membrane-anchored plakoglobins have multiple mechanisms of action in Wnt signaling.
- A comparative evaluation of beta-catenin and plakoglobin signaling activity.
- PPARs and the complex journey to obesity.
- PPARs and LXRs: atherosclerosis goes nuclear.
- A nuclear strike against Listeria--the evolving life of LXR.
- Pregnane X receptor prevents hepatorenal toxicity from cholesterol metabolites.
- A Nuclear Receptor Atlas: macrophage activation.
- Peroxisome proliferator-activated receptors and liver X receptors in atherosclerosis and immunity.
- PPAR delta: a dagger in the heart of the metabolic syndrome.
- Nuclear receptor ERR alpha and coactivator PGC-1 beta are effectors of IFN-gamma-induced host defense.
- PPARdelta regulates multiple proinflammatory pathways to suppress atherosclerosis.
- Bcl-6 and NF-kappaB cistromes mediate opposing regulation of the innate immune response.
- NCoR1 is a conserved physiological modulator of muscle mass and oxidative function.
- Cryptochromes mediate rhythmic repression of the glucocorticoid receptor.
- The LPS2 mutation in TRIF is atheroprotective in hyperlipidemic low density lipoprotein receptor knockout mice.
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