Dr. Mary E Choi M.D.
Nephrologist (Kidney Specialist) | Nephrology
3550 Terrace St Pittsburgh PA, 15213About
Dr. Mary Choi is a nephrologist practicing in Boston, MA. Dr. Choi specializes in the care and treatment of the kidneys. As a nephrologist, Dr. Choi most typically treats conditions like kidney stones, chronic kidney disease, acute renal failure, polycystuc kidney disease, high blood pressure and more. Nephrologists are also experts on kidney transplantation and dialysis. They are usually referred to by primary care physicians for problems related to the kidneys, and while they can perform tests to diagnose kidney disorders, they do not perform surgeries.
Education and Training
Univ of Ks Sch of Med, Kansas City Ks 1984
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Requirement of mitogen-activated protein kinase kinase 3 (MKK3) for activation of p38alpha and p38delta MAPK isoforms by TGF-beta 1 in murine mesangial cells.
- TGF-beta1 stimulates HO-1 via the p38 mitogen-activated protein kinase in A549 pulmonary epithelial cells.
- Transforming growth factor-beta1 stimulates vascular endothelial growth factor 164 via mitogen-activated protein kinase kinase 3-p38alpha and p38delta mitogen-activated protein kinase-dependent pathway in murine mesangial cells.
- Caveolin-1 expression by means of p38beta mitogen-activated protein kinase mediates the antiproliferative effect of carbon monoxide.
- Stimulation of cyclooxygenase-2 expression by bone-derived transforming growth factor-beta enhances bone metastases in breast cancer.
- TGF-beta-activated kinase 1 and TAK1-binding protein 1 cooperate to mediate TGF-beta1-induced MKK3-p38 MAPK activation and stimulation of type I collagen.
- Protective effects of low-dose carbon monoxide against renal fibrosis induced by unilateral ureteral obstruction.
- Protein phosphatase 2A is a negative regulator of transforming growth factor-beta1-induced TAK1 activation in mesangial cells.
- BAT3 interacts with transforming growth factor-beta (TGF-beta) receptors and enhances TGF-beta1-induced type I collagen expression in mesangial cells.
- The not-so-sweet side of fructose.
- Transforming growth factor-beta (TGF-beta1) activates TAK1 via TAB1-mediated autophosphorylation, independent of TGF-beta receptor kinase activity in mesangial cells.
- TGF-{beta}1 protects against mesangial cell apoptosis via induction of autophagy.
- REIN on obesity, proteinuria and CKD.
- Autophagy promotes intracellular degradation of type I collagen induced by transforming growth factor (TGF)-β1.
- TGF-β signaling via TAK1 pathway: role in kidney fibrosis.
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