Dr. Robert C Harland MD
Surgeon
600 Moye Blvd Ecu Physicians Greenville NC, 27834About
Dr. Robert Harland is a general surgeon practicing in Greenville, NC. Dr. Harland specializes in abdominal contents including the esophagus, stomach, liver, gallbladder, pancreas and often thyroid glands. General surgeons are able to deal with almost any surgical or critical care emergency, also involving the skin or soft tissue trauma. Dr. Harland provides quality surgical service for gravely ill or injured patients and is able to respond quickly due to knowledge of various surgical procedures.
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
SurgeryAmerican Board of SurgeryABS
Provider Details
Expert Publications
Data provided by the National Library of Medicine- P-Selectin mediates reperfusion injury through neutrophil and platelet sequestration in the warm ischemic mouse liver.
- Bile acids in xenogeneic ex-vivo liver perfusion: function of xenoperfused livers and compatibility with human bile salts and porcine livers.
- Case report: robot-assisted laparoscopic pyeloureterostomy in a transplanted kidney with ureteral stricture.
- Hepato-spleno-mesenteric trunk.
- Probabilistic risk assessment of accidental ABO-incompatible thoracic organ transplantation before and after 2003.
- Fifteen-year follow-up of transplantation of a cadaveric polycystic kidney: a case report.
- Irradiation for xenogeneic transplantation.
- A report of the epidemiology of de novo donor-specific anti-HLA antibodies (DSA) in "low-risk" renal transplant recipients.
- Significant prolongation of hamster liver transplant survival in Lewis rats by total-lymphoid irradiation, cyclosporine, and splenectomy.
- Incidence and impact of de novo donor-specific alloantibody in primary renal allografts.
- The role of immunoglobulin-G subclasses and C1q in de novo HLA-DQ donor-specific antibody kidney transplantation outcomes.
- Trends and characteristics in early glomerular filtration rate decline after posttransplantation alloantibody appearance.
- Higher risk of kidney graft failure in the presence of anti-angiotensin II type-1 receptor antibodies.
- Impact of IgM and IgG3 anti-HLA alloantibodies in primary renal allograft recipients.
- Changes in successive measures of de novo donor-specific anti-human leukocyte antigen antibodies intensity and the development of allograft dysfunction.
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