Dr. David H Van thiel MD
Hepatologist | Hepatology
3501 Harlem Ave Berwyn IL, 60402About
Dr. David Van thiel is a hepatologist practicing in Milwaukee, WI. Dr. Van thiel specializes in the study of body parts such as the liver, the biliary tree, the gallbladder and the pancreas. Hepatologists manage disorders in these areas and are specifically known for treating viral hepatitis. Patients who have received liver transplants also follow up with hepatologists after their procedure.
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Expert Publications
Data provided by the National Library of Medicine- Kupffer cell expression of CD40L (CD154) in human chronic liver allograft rejection.
- Gender influences cold preservation-reoxygenation injury in the liver.
- Sodium salicylate protects hepatocytes but not endothelial cells during reperfusion after cold ischemia.
- CD40 upregulation in TCR alpha/beta+ CD68+ cells and parenchymal CD40L induction and associated with NF-kappa B activation in chronic rejecting human renal allografts.
- Ischemia-reperfusion injury in rat fatty liver: role of nutritional status.
- CD40 expression on graft infiltrates and parenchymal CD154 (CD40L) induction in human chronic renal allograft rejection.
- Conventional care of fulminant hepatic failure: medical and surgical aspects.
- The diagnosis and treatment of Clostridium difficile in antibiotic-associated diarrhea.
- Interferon therapy of Turkish patients with chronic hepatitis B virus infection.
- Immune status of individuals with differing clinical courses of HBV infection.
- Hyperprolactinemia and thyrotropin-releasing factor (TRH) responses in men with alcoholic liver disease.
- Hyperprolactinemia and thyrotropin-releasing factor (TRH) responses in men with alcoholic liver disease.
- Can hepatitis B core antibody positive livers be used safely for transplantation: hepatitis B virus detection in the liver of individuals who are hepatitis B core antibody positive.
- Regional differences on production of chemokines in gastric mucosa between Helicobacter pylori-positive duodenal ulcer and gastric ulcer.
- Oxidative DNA damage in circulating leukocytes occurs as an early event in chronic HCV infection.
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