Dr. Ja Gu Kang MD
Internist
60 07 43rd Ave Woodside NY, 11377Rating
3/5
About
Dr. Ja Kang is an internist practicing in Woodside, NY. Dr. Kang specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Kang provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
Kyongpook Natl Univ, Coll of Med, Taegu, So Korea 1962
Kyungpuk National University / College of Medicine 1962
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- RsrA, an anti-sigma factor regulated by redox change.
- Isolation and identification of antifungal N-butylbenzenesulphonamide produced by Pseudomonas sp. AB2.
- Chromophore-apoprotein interactions in Synechocystis sp. PCC6803 phytochrome Cph1.
- A second photochromic bacteriophytochrome from Synechocystis sp. PCC 6803: spectral analysis and down-regulation by light.
- Drosophila Mediator complex is broadly utilized by diverse gene-specific transcription factors at different types of core promoters.
- Light and brassinosteroid signals are integrated via a dark-induced small G protein in etiolated seedling growth.
- Application technique and irrigation method affect imidacloprid control of silverleaf whiteflies (Homoptera: Aleyrodidae) on poinsettias.
- Histone tails modulate nucleosome mobility and regulate ATP-dependent nucleosome sliding by NURF.
- The relationship between inhalational anesthetic requirements and the severity of liver disease in liver transplant recipients according to three phases of liver transplantation.
- Randomized controlled trial to investigate the effects of a newly developed formulation of phentermine diffuse-controlled release for obesity.
- Effects of all-trans retinoic acid on sodium/iodide symporter and CCAAT/enhancer-binding protein-homologous protein under condition of endoplasmic reticulum stress in FRTL5 thyroid cells.
- Alpha-lipoic acid inhibits endoplasmic reticulum stress-induced cell death through PI3K/Akt signaling pathway in FRTL5 thyroid cells.
- CCAAT/Enhancer-binding protein-homologous protein sensitizes to SU5416 by modulating p21 and PI3K/Akt signal pathway in FRO anaplastic thyroid carcinoma cells.
- Inhibition of p21 and Akt potentiates SU6656-induced caspase-independent cell death in FRO anaplastic thyroid carcinoma cells.
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