Dr. Lawrence Fisher Brass MD
Oncologist | Medical Oncology
3400 Spruce St 15 Penn Tower Philadelphia PA, 19104About
Dr. Lawrence Brass is an oncologist practicing in Philadelphia, PA. Dr. Brass specializes in the care and treatment of patients with cancer. As an oncologist, Dr. Brass manages and oversees the treatment of a cancer patient after he or she has been diagnosed with the disease. Oncologists will care for their patients throughout the course of the disease. Types of oncologists include medical oncologists, surgical oncologists, radiation oncologists, gynecologic oncologists, pediatric oncologists and hematologist oncologists.
Education and Training
Case Western Reserve University School of Medicine 1977
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Megakaryocyte precursors, megakaryocytes and platelets express the HIV co-receptor CXCR4 on their surface: determination of response to stromal-derived factor-1 by megakaryocytes and platelets.
- PAR1 activation initiates integrin engagement and outside-in signalling in megakaryoblastic CHRF-288 cells.
- More pieces of the platelet activation puzzle slide into place.
- CXCR4 on human endothelial cells can serve as both a mediator of biological responses and as a receptor for HIV-2.
- Thrombin responses in human endothelial cells. Contributions from receptors other than PAR1 include the transactivation of PAR2 by thrombin-cleaved PAR1.
- The alpha subunits of Gz and Gi interact with the eyes absent transcription cofactor Eya2, preventing its interaction with the six class of homeodomain-containing proteins.
- Loss of signaling through the G protein, Gz, results in abnormal platelet activation and altered responses to psychoactive drugs.
- Neutrophil proteases can inactivate human PAR3 and abolish the co-receptor function of PAR3 on murine platelets.
- Protease activated receptors: theme and variations.
- Evaluation of the substrate specificity of human mast cell tryptase beta I and
- Gene induction by coagulation factor Xa is mediated by activation of
- A comparison of the binding to polynucleotides of complementary and noncomplementary oligonucleotides.
- Extracellular signal-regulated kinases and g protein-coupled receptors in megakaryocytic human erythroleukemia cells: selective activation, differential regulation, and dissociation from mitogenesis.
- Activation of Rap1B by G(i) family members in platelets.
- Interactions between Eph kinases and ephrins provide a mechanism to support platelet aggregation once cell-to-cell contact has occurred.
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