Dr. Harold S Sacks M.D.
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
6027 Walnut Grove Rd Suite 307 Memphis TN, 38120About
Dr. Harold Sacks practices Endocrinology in Memphis, TN. Dr. Sacks specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Sacks examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ Of Cape Town- Fac Of Med- Cape Town- So Africa 1967
University of Cape Town / Faculty of Health Sciences 1967
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Human epicardial adipose tissue: a review.
- Weight loss in obesity reduces epicardial fat thickness; so what?
- The importance of brown adipose tissue.
- Epicardial fat gene expression after aerobic exercise training in pigs with coronary atherosclerosis: relationship to visceral and subcutaneous fat.
- Human epicardial fat: what is new and what is missing?
- Adult epicardial fat exhibits beige features.
- Exercise training does not increase muscle FNDC5 protein or mRNA expression in pigs.
- Epicardial adipose excision slows the progression of porcine coronary atherosclerosis.
- Adipocyte Browning and Higher Mitochondrial Function in Periadrenal But Not SC Fat in Pheochromocytoma.
- 'Browning' the cardiac and peri-vascular adipose tissues to modulate cardiovascular risk.
- Loss of UCP1 exacerbates Western diet-induced glycemic dysregulation independent of changes in body weight in female mice.
- Epicardial Adipose Tissue Removal Potentiates Outward Remodeling and Arrests Coronary Atherogenesis.
- Deletion of UCP1 enhances ex vivo aortic vasomotor function in female but not male mice despite similar susceptibility to metabolic dysfunction.
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