Edward Gerard Lakatta M.D.
Cardiologist | Cardiovascular Disease
5600 Nathan Shock Dr Baltimore MD, 21224About
Dr. Edward Lakatta is a cardiologist practicing in Baltimore, MD. Dr. Lakatta specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Lakatta also practices preventative medicine, helping patients maintain a heart-healthy life.
Education and Training
Georgetown Univ Sch of Med, Washington Dc 1970
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Cardiovascular Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Myocardial retinoid X receptor, thyroid hormone receptor, and myosin heavy chain gene expression in the rat during adult aging.
- beta2-adrenergic cAMP signaling is uncoupled from phosphorylation of cytoplasmic proteins in canine heart.
- Cardiovascular aging research: the next horizons.
- Bcl-2 and Bcl-X(L) block thapsigargin-induced nitric oxide generation, c-Jun NH(2)-terminal kinase activity, and apoptosis.
- Cardiac-specific overexpression of the alpha(1) subunit of the L-type voltage-dependent Ca(2+) channel in transgenic mice. Loss of isoproterenol-induced contraction.
- G(i) protein-mediated functional compartmentalization of cardiac beta(2)-adrenergic signaling.
- Spontaneous beta(2)-adrenergic signaling fails to modulate L-type Ca(2+) current in mouse ventricular myocytes.
- Hemodynamic effects of unloading the old heart.
- Recent advances in cardiac beta(2)-adrenergic signal transduction.
- Constitutive beta2-adrenergic signalling enhances sarcoplasmic reticulum Ca2+ cycling to augment contraction in mouse heart.
- Cicletanine reverses vasoconstriction induced by the endogenous sodium pump ligand, marinobufagenin, via a protein kinase C dependent mechanism.
- Culture and adenoviral infection of adult mouse cardiac myocytes: methods for cellular genetic physiology.
- Induction of angiogenesis by cationic lipid-mediated VEGF165 gene transfer in the rabbit ischemic hindlimb model.
- Cardiovascular aging in health.
- Adenovirus-mediated VEGF(121) gene transfer stimulates angiogenesis in normoperfused skeletal muscle and preserves tissue perfusion after induction of ischemia.
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