Dr. Cory Dalton Dunn MD
Pathologist | Anatomic Pathology & Clinical Pathology
1801 16th St Greeley CO, 80631About
Dr. Cory Dunn is a pathologist practicing in Greeley, CO. Dr. Dunn is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Dunn can help your primary care doctor make a diagnosis about your medical condition. Dr. Dunn may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Education and Training
St Louis Univ Sch of Med, St Louis Mo 1984
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Protein import into and across the mitochondrial inner membrane: role of the TIM23 and TIM22 translocons.
- Suppression of a defect in mitochondrial protein import identifies cytosolic proteins required for viability of yeast cells lacking mitochondrial DNA.
- Bacterial and host factors involved in the major histocompatibility complex class Ib-restricted presentation of Salmonella Hsp 60: novel pathway.
- Mitochondrial building blocks.
- A genomewide screen for petite-negative yeast strains yields a new subunit of the i-AAA protease complex.
- Ups1p, a conserved intermembrane space protein, regulates mitochondrial shape and alternative topogenesis of Mgm1p.
- Awaking TIM22, a dynamic ligand-gated channel for protein insertion in the mitochondrial inner membrane.
- Mgr3p and Mgr1p are adaptors for the mitochondrial i-AAA protease complex.
- Running on empty: does mitochondrial DNA mutation limit replicative lifespan in yeast?: Mutations that increase the division rate of cells lacking mitochondrial DNA also extend replicative lifespan in Saccharomyces cerevisiae.
- Defects associated with mitochondrial DNA damage can be mitigated by increased vacuolar pH in Saccharomyces cerevisiae.
- Deletion of conserved protein phosphatases reverses defects associated with mitochondrial DNA damage in Saccharomyces cerevisiae.
- Activation of the pleiotropic drug resistance pathway can promote mitochondrial DNA retention by fusion-defective mitochondria in Saccharomyces cerevisiae.
- Mutation of the mitochondrial large ribosomal RNA can provide pentamidine resistance to Saccharomyces cerevisiae.
- Reduced Glucose Sensation Can Increase the Fitness of Saccharomyces cerevisiae Lacking Mitochondrial DNA.
- Evidence for Amino Acid Snorkeling from a High-Resolution, In Vivo Analysis of Fis1 Tail-Anchor Insertion at the Mitochondrial Outer Membrane.
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