Dr. William Michael Pandak M.D.
Gastroenterologist | Gastroenterology
Veterans Affairs Medical Center And Va Commonwealth U Division Of Gastroen Richmond VA, 23249About
Dr. William Pandak is a gastroenterologist practicing in Richmond, VA. Dr. Pandak specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Pandak performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Education and Training
Va Commonwealth Univ, Med Coll of Va Sch of Med, Richmond Va 1983
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Gastroenterology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Regulation of sterol 12alpha-hydroxylase and cholic acid biosynthesis in the rat.
- Regulation of multidrug resistance 2 P-glycoprotein expression by bile salts in rats and in primary cultures of rat hepatocytes.
- Expression of sterol 12alpha-hydroxylase alters bile acid pool composition in primary rat hepatocytes and in vivo.
- LXR alpha is the dominant regulator of CYP7A1 transcription.
- Short course of omeprazole: a better first diagnostic approach to noncardiac chest pain than endoscopy, manometry, or 24-hour esophageal pH monitoring.
- Transport of cholesterol into mitochondria is rate-limiting for bile acid synthesis via the alternative pathway in primary rat hepatocytes.
- Regulation of oxysterol 7alpha-hydroxylase (CYP7B1) in the rat.
- Indinavir alters sterol and fatty acid homeostatic mechanisms in primary rat hepatocytes by increasing levels of activated sterol regulatory element-binding proteins and decreasing cholesterol 7alpha-hydroxylase mRNA levels.
- The role of alpha1-fetoprotein transcription factor/LRH-1 in bile acid biosynthesis: a known nuclear receptor activator that can act as a suppressor of bile acid biosynthesis.
- Hormonal regulation of cholesterol 7 alpha-hydroxylase mRNA levels and transcriptional activity in primary rat hepatocyte cultures.
- Effect of increasing the expression of cholesterol transporters (StAR, MLN64, and SCP-2) on bile acid synthesis.
- Overexpression of cholesterol transporter StAR increases in vivo rates of bile acid synthesis in the rat and mouse.
- Detection of the steroidogenic acute regulatory protein, StAR, in human liver cells.
- Human StarD5, a cytosolic StAR-related lipid binding protein.
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