Dr. Thomas Gerald Beach M.D.
Neuropathologist | Neuropathology
10515 W Santa Fe Dr Sun City AZ, 85351About
Dr. Thomas Beach is a Neuropsychiatrist practicing in Sun City, AZ. Dr. Beach studies, evaluates, diagnoses, and treats mental disorders attributable to diseases of the nervous system. Neuropsychiatrists are trained to treat disorders occurring in patients due to irritability, attention deficit disorder, epilepsy, and many other conditions.
Education and Training
Univ Of British Columbia- Fac Of Med- Vancouver- Bc- Canada 1985
University of British Columbia Faculty of Medicine 1985
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Increased expression of the urokinase plasminogen-activator receptor in amyloid beta peptide-treated human brain microglia and in AD brains.
- Muscarinic agonists as preventative therapy for Alzheimer's disease.
- Cortical and leptomeningeal cerebrovascular amyloid and white matter pathology in Alzheimer's disease.
- Hippocampal sclerosis dementia with tauopathy.
- Circle of willis atherosclerosis is a risk factor for sporadic Alzheimer's disease.
- Thiorphan-induced neprilysin inhibition raises amyloid beta levels in rabbit cortex and cerebrospinal fluid.
- Substantia nigra Marinesco bodies are associated with decreased striatal expression of dopaminergic markers.
- Atherosclerosis, vascular amyloidosis and brain hypoperfusion in the pathogenesis of sporadic Alzheimer's disease.
- Atherosclerosis of cerebral arteries in Alzheimer disease.
- An association with great implications: vascular pathology and Alzheimer disease.
- Amyloid-beta peptide remnants in AN-1792-immunized Alzheimer's disease patients: a biochemical analysis.
- Interaction of cardiovascular disease and neurodegeneration: transcranial Doppler ultrasonography and Alzheimer's disease.
- Mutations in progranulin are a major cause of ubiquitin-positive frontotemporal lobar degeneration.
- Pathologic and nicotinic receptor binding differences between mild cognitive impairment, Alzheimer disease, and normal aging.
- Alterations in immunological and neurological gene expression patterns in Alzheimer's disease tissues.
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