Bradley R Pike PT
Physical Therapist
314 E 1st St East Syracuse NY, 13057About
Bradley Pike is a physical therapist practicing in East Syracuse, NY. Bradley Pike specializes in physical treatment to help a patient reduce pain, restore mobility, rehabilitate an injury, or increase movement and overall function. As a physical therapist, Bradley Pike can treat multiple conditions with exercises, ultrasound, electrical stimulation, joint mobilization, heat, ice, massage, laser or light therapy and more. Bradley Pike will create a treatment plan based on the patients specific injury or condition, and might target a specific body part or body system based on the individual.
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Expert Publications
Data provided by the National Library of Medicine- Temporal profile of apoptotic-like changes in neurons and astrocytes following controlled cortical impact injury in the rat.
- Casein zymogram assessment of mu-calpain and m-calpain activity after traumatic brain injury in the rat in vivo.
- Concurrent assessment of calpain and caspase-3 activity by means of western blots of protease-specific spectrin breakdown products.
- TNF-alpha stimulates caspase-3 activation and apoptotic cell death in primary septo-hippocampal cultures.
- Temporal and spatial profile of caspase 8 expression and proteolysis after experimental traumatic brain injury.
- Accumulation of non-erythroid alpha II-spectrin and calpain-cleaved alpha II-spectrin breakdown products in cerebrospinal fluid after traumatic brain injury in rats.
- Concurrent assessment of calpain and caspase-3 activation after oxygen-glucose deprivation in primary septo-hippocampal cultures.
- Systemic administration of a calpain inhibitor reduces behavioral deficits and blood-brain barrier permeability changes after experimental subarachnoid hemorrhage in the rat.
- Effects of injury severity on regional and temporal mRNA expression levels of calpains and caspases after traumatic brain injury in rats.
- Impaired gustatory neophobia following traumatic brain injury in rats.
- The effect of postinjury kindled seizures on cognitive performance of traumatically brain-injured rats.
- Post-injury administration of BIBN 99, a selective muscarinic M2 receptor antagonist, improves cognitive performance following traumatic brain injury in rats.
- Cognitive impairment following traumatic brain injury: the effect of pre- and post-injury administration of scopolamine and MK-801.
- Traumatic brain injury enhances the amnesic effect of an NMDA antagonist in rats.
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