Dr. Kenneth Robert Cooke MD
Hematologist (Pediatric) | Pediatric Hematology-Oncology
Crb1 Rm 207 1650 Orleans Street, Baltimore MD, 21287Rating
5/5
About
Dr. Kenneth Cooke is a pediatric hematologist practicing in Baltimore, MD. Dr. Cooke specializes in treating children that have a blood disease or cancer. Such blood diseases include disorders of red blood cells, white blood cells and/or platelets. The types of cancers that Dr. Cooke treats include leukemias, lymphomas and certain tumors. Dr. Cooke can also treat bleeding disorders in children. Pediatric hematologists can be found in childrens hospitals, community hospitals, university medical centers and more.
Education and Training
Umdnj-Robt W Johnson Med Sch- New Brunswick Nj 1990
University of Medicine And Dentistry of New Jersey / Robert Wood Johns 1990
University of Medicine And Dentistry of New Jersey / Robert Wood Johnson Med 1990
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Etanercept (Enbrel) administration for idiopathic pneumonia syndrome after allogeneic hematopoietic stem cell transplantation.
- The pathophysiology of acute graft-versus-host disease.
- A critical role for CCR2/MCP-1 interactions in the development of idiopathic pneumonia syndrome after allogeneic bone marrow transplantation.
- Donor-derived TNF-alpha regulates pulmonary chemokine expression and the development of idiopathic pneumonia syndrome after allogeneic bone marrow transplantation.
- Induction of heme oxygenase-1 before conditioning results in improved survival
- Blockade of CXCR3 receptor:ligand interactions reduces leukocyte recruitment to the lung and the severity of experimental idiopathic pneumonia syndrome.
- A role for tumor necrosis factor-alpha-mediated endothelial apoptosis in the development of experimental idiopathic pneumonia syndrome.
- Donor T-cell production of RANTES significantly contributes to the development of idiopathic pneumonia syndrome after allogeneic stem cell transplantation.
- A role for CD54 (intercellular adhesion molecule-1) in leukocyte recruitment to the lung during the development of experimental idiopathic pneumonia syndrome.
- Acute lung injury after allogeneic stem cell transplantation: from the clinic, to the bench and back again.
- The lung as a target organ of graft-versus-host disease.
- The absence of donor-derived IL-13 exacerbates the severity of acute graft-versus-host disease following allogeneic bone marrow transplantation.
- Bronchiolitis obliterans syndrome (BOS), bronchiolitis obliterans organizing pneumonia (BOOP), and other late-onset noninfectious pulmonary complications following allogeneic hematopoietic stem cell transplantation.
- CCR1/CCL5 (RANTES) receptor-ligand interactions modulate allogeneic T-cell responses and graft-versus-host disease following stem-cell transplantation.
- The contribution of endothelial activation and injury to end-organ toxicity following allogeneic hematopoietic stem cell transplantation.
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