Dr. Laura A Arbogast DO
Internist
385 N Lapeer Rd Oxford MI, 48371About
Dr. Laura Arbogast is an internist practicing in Oxford, MI. Dr. Arbogast specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Arbogast provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
Michigan State University College of Osteopathic Medicine 2002
Provider Details
Expert Publications
Data provided by the National Library of Medicine- 3,5 cyclic adenosine monophosphate mediates the salmon calcitonin-induced increase in hypothalamic tyrosine hydroxylase activity.
- Pituitary hormone gene expression and secretion in human growth hormone-releasing hormone transgenic mice: focus on lactotroph function.
- Estradiol attenuates the forskolin-induced increase in hypothalamic tyrosine hydroxylase activity.
- Calcitonin inhibition of prolactin secretion in lactating rats: mechanism of action.
- Calmodulin and a cyclic nucleotide-dependent protein kinase facilitate the prolactin-induced increase in tyrosine hydroxylase activity in tuberoinfundibular dopaminergic neurons.
- Mechanisms of tyrosine hydroxylase regulation during pregnancy: evidence for protein dephosphorylation during the prolactin surges.
- Ontogeny of tyrosine hydroxylase mRNA signal levels in central dopaminergic neurons: development of a gender difference in the arcuate nuclei.
- Hyperprolactinemia increases and hypoprolactinemia decreases tyrosine hydroxylase messenger ribonucleic acid levels in the arcuate nuclei, but not the substantia nigra or zona incerta.
- The preovulatory prolactin surge is prolonged by a progesterone-dependent dopaminergic mechanism.
- Tyrosine hydroxylase messenger RNA in the hypothalamus, substantia nigra and adrenal medulla of old female rats.
- Sex-related alterations in hypothalamic tyrosine hydroxylase after neonatal monosodium glutamate treatment.
- Gene expression profiles of intracellular and membrane progesterone receptor isoforms in the mediobasal hypothalamus during pro-oestrus.
- Mu and kappa opioid receptor expression in the mediobasal hypothalamus and effectiveness of selective antagonists on prolactin release during lactation.
- Hypoprolactinemia decreases tyrosine hydroxylase activity in the tuberoinfundibular dopaminergic neurons acutely by protein dephosphorylation and chronically by changes in gene expression.
- Rat placental lactogen-I abolishes nocturnal prolactin surges in the pregnant rat.
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