Polly J Ferguson
Rheumatologist (Pediatric) | Pediatric Rheumatology200 Hawkins Dr Iowa City IA, 52242
Dr. Polly Ferguson practices Pediatric Rheumatology in Iowa City, IA. Pediatric rheumatologists treat child patients who suffer from pain in the musculoskeletal system, have symptoms of arthritis, or an autoimmune disorder. Dr. Ferguson evaluates and treats a variety of joint, muscle, and bone disorders; including autoimmune disorders, such as lupus, juvenile rheumatoid arthritis, scleroderma, Kawasaki disease, post-infectious arthritis, chronic vasculitis, and inflammatory disorders of the muscle, eye, or other organs. Dr. Ferguson also is trained in the evaluation of prolonged fever, unexplained complaints of chronic musculoskeletal pain, weakness, poor appetite, fatigue, and/or loss of function or skills.
Education and Training
Univ Of Ia Coll Of Med- Iowa City Ia 1990
Baylor College of Medicine, Houston, Texas 1990
PediatricsAmerican Board of PediatricsABP- Pediatric Rheumatology
Expert PublicationsData provided by the National Library of Medicine
- Shrinking lung syndrome in a 14-year-old boy with systemic lupus erythematosus.
- A splice site mutation confirms the role of LPIN2 in Majeed syndrome.
- Chronic recurrent multifocal osteomyelitis: a concise review and genetic update.
- Primary multifocal osseous lymphoma in a child.
- Papular xanthomas and erosive arthritis in a 3 year old girl, is this a new MRH variant?
- Current understanding of the pathogenesis and management of chronic recurrent multifocal osteomyelitis.
- Efficacy of anti-IL-1 treatment in Majeed syndrome.
- Chronic nonbacterial osteomyelitis in a child with previous juvenile dermatomyositis.
- Genome-wide association mapping in dogs enables identification of the homeobox gene, NKX2-8, as a genetic component of neural tube defects in humans.
- Autoinflammatory bone disorders: update on immunologic abnormalities and clues about possible triggers.
- Exome sequencing reveals RAG1 mutations in a child with autoimmunity and sterile chronic multifocal osteomyelitis evolving into disseminated granulomatous disease.
- Autoinflammatory bone diseases.
- Inflammasome-independent IL-1β mediates autoinflammatory disease in Pstpip2-deficient mice.
- The F-BAR protein PSTPIP1 controls extracellular matrix degradation and filopodia formation in macrophages.
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