Dr. Roger Scott Rittmaster M.D.
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
42 William Glen Dr Camden ME, 04843Rating
2/5
About
Dr. Roger Rittmaster practices Endocrinology in Camden, ME. Dr. Rittmaster specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Rittmaster examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Tufts Univ Sch of Med, Boston Ma 1976
Tufts University School of Medicine 1976
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Antiandrogen treatment of polycystic ovary syndrome.
- Androgen-induced regrowth in the castrated rat ventral prostate: role of 5alpha-reductase.
- Prostatic involution in men taking finasteride is associated with elevated levels of insulin-like growth factor-binding proteins (IGFBPs)-2, -4, and -5 .
- Dihydrotestosterone and the concept of 5alpha-reductase inhibition in human benign prostatic hyperplasia.
- Enhancement of bone mass in osteoporotic women with parathyroid hormone followed by alendronate.
- Dihydrotestosterone and the concept of 5alpha-reductase inhibition in human
- [Dihydrotestosterone and the role of 5 alpha-reductase inhibitors in benign prostatic hyperplasia].
- 5alpha-reductase type 1 immunostaining is enhanced in some prostate cancers compared with benign prostatic hyperplasia epithelium.
- Dutasteride, the dual 5alpha-reductase inhibitor, inhibits androgen action and promotes cell death in the LNCaP prostate cancer cell line.
- Effect of the dual 5alpha-reductase inhibitor dutasteride on markers of tumor regression in prostate cancer.
- Hyperandrogenism--what is normal?
- Effect of dutasteride on the detection of prostate cancer in men with benign prostatic hyperplasia.
- Differential alterations in 5alpha-reductase type 1 and type 2 levels during development and progression of prostate cancer.
- Relationship among serum testosterone, sexual function, and response to treatment in men receiving dutasteride for benign prostatic hyperplasia.
- DMP1 mutations in autosomal recessive hypophosphatemia implicate a bone matrix protein in the regulation of phosphate homeostasis.
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