Dr. David T Woodley MD
Dermatologist
1520 San Pablo St 1000a Los Angeles CA, 90033About
Dr. David Woodley is a dermatologist practicing in Los Angeles, CA. Dr. Woodley specializes in skin care. Dermatologists evaluate and manage both common and uncommon skin conditions. These conditions include acne, psoriasis, warts, skin infections, atopic dermatitis, herpes simplex and more. Dermatologists are also experts in more complex skin diseases like impetigo, hidradenitis and milaria. Dr. Woodley diagnoses skin problems and develops unique treatments plans for each individual patient.
Education and Training
Univ of Mo, Columbia Sch of Med, Columbia Mo 1972
University of Pennsylvania, PA 1973
University of British Columbia Faculty of Medicine 1972
Board Certification
DermatologyAmerican Board of DermatologyABD
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Alpha 2 beta 1 integrin mediates dermal fibroblast attachment to type VII collagen via a 158-amino-acid segment of the NC1 domain.
- Matrix localization of tissue factor pathway inhibitor-2/matrix-associated serine protease inhibitor (TFPI-2/MSPI) involves arginine-mediated ionic interactions with heparin and dermatan sulfate: heparin accelerates the activity of TFPI-2/MSPI toward
- Thalidomide increases human keratinocyte migration and proliferation.
- Induction of keratinocyte IL-8 expression and secretion by IgG autoantibodies as a novel mechanism of epidermal neutrophil recruitment in a pemphigus variant.
- Development and characterization of a recombinant truncated type VII collagen "minigene". Implication for gene therapy of dystrophic epidermolysis bullosa.
- Nckbeta adapter regulates actin polymerization in NIH 3T3 fibroblasts in response to platelet-derived growth factor bb.
- Prokaryotic expression, purification, and reconstitution of biological activities (Antiprotease, antitumor, and heparin-binding) for tissue factor pathway inhibitor-2.
- Colony-stimulating factor-1 receptor utilizes multiple signaling pathways to induce cyclin D2 expression.
- Autoantibodies to type VII collagen have heterogeneous subclass and light chain compositions and their complement-activating capacities do not correlate with the inflammatory clinical phenotype.
- The carboxyl terminus of type VII collagen mediates antiparallel dimer formation and constitutes a new antigenic epitope for epidermolysis Bullosa acquisita autoantibodies.
- Phosphorylation of focal adhesion kinase (pp125(FAK)) is increased in human keratinocytes induced to migrate by extracellular matrices.
- Nck/Dock: an adapter between cell surface receptors and the actin cytoskeleton.
- The recombinant expression of full-length type VII collagen and characterization of molecular mechanisms underlying dystrophic epidermolysis bullosa.
- Epidermolysis bullosa acquisita: update and review.
- An important role for protein kinase C-delta in human keratinocyte migration on dermal collagen.
Treatments
- Acne
- Dermatitis
- Moles
- Seborrheic Dermatitis
- Actinic Keratosis
- Skin Cancer
- Birthmark
- Allergic Reaction
- Seborrheic Keratosis
- Peripheral Artery Disease (pad)
Dr. David T Woodley MD's Practice location
Los Angeles, CA 90033Get Direction
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