Dr. Mary Margaret Sugrue MD
Hematologist (Pediatric) | Pediatric Hematology-Oncology
166 Pine St Garden City NY, 11530About
Dr. Mary Sugrue is a pediatric hematologist practicing in Garden City, NY. Dr. Sugrue specializes in treating children that have a blood disease or cancer. Such blood diseases include disorders of red blood cells, white blood cells and/or platelets. The types of cancers that Dr. Sugrue treats include leukemias, lymphomas and certain tumors. Dr. Sugrue can also treat bleeding disorders in children. Pediatric hematologists can be found in childrens hospitals, community hospitals, university medical centers and more.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- p21Waf1/Cip1/Sdi1 induces permanent growth arrest with markers of replicative senescence in human tumor cells lacking functional p53.
- Reduced mitochondrial membrane potential and altered responsiveness of a mitochondrial membrane megachannel in p53-induced senescence.
- Glyceraldehyde-3-phosphate dehydrogenase in neurodegeneration and apoptosis signaling.
- Mitochondrial membrane potential in aging cells.
- Immunocytochemical localization of the neuron-specific form of the c-src gene product, pp60c-src(+), in rat brain.
- Bevacizumab beyond first progression is associated with prolonged overall survival in metastatic colorectal cancer: results from a large observational cohort study (BRiTE).
- Clinical outcomes associated with bevacizumab-containing treatment of metastatic
- p53-induced growth arrest is regulated by the mitochondrial SirT3 deacetylase.
- Validation of the IPSS-R in lenalidomide-treated, lower-risk myelodysplastic syndrome patients with del(5q).
- Real-world analysis of the Celgene Global Drug Safety database: early discontinuation of lenalidomide in patients with myelodysplastic syndromes due to non-serious rash.
- Connect MDS/AML: design of the myelodysplastic syndromes and acute myeloid leukemia disease registry, a prospective observational cohort study.
- Azacitidine in Lower-Risk Myelodysplastic Syndromes: A Meta-Analysis of Data from Prospective Studies.
- Wild-type p53 triggers a rapid senescence program in human tumor cells lacking functional p53.
- Inhibition of tumor cell growth by RTP/rit42 and its responsiveness to p53 and DNA damage.
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