Dr. Evan Dale Abel MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
615 Arapeen Dr 100 Salt Lake City UT, 84108About
Dr. Evan Abel practices Endocrinology in Salt Lake City, UT. Dr. Abel specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Abel examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Univ Of West Indies- Fac Med Sci- Kingston- Jamaica 1985
Faculty of Medical Sciences, University of The West Indies (Jamaica) 1985
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology, Diabetes and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Insulin signaling coordinately regulates cardiac size, metabolism, and contractile protein isoform expression.
- Akt signaling mediates postnatal heart growth in response to insulin and nutritional status.
- Gender-dependent attenuation of cardiac potassium currents in type 2 diabetic db/db mice.
- Glucose transport in the heart.
- Targeted deletion of BMK1/ERK5 in adult mice perturbs vascular integrity and leads to endothelial failure.
- Regulation of insulin-responsive aminopeptidase expression and targeting in the insulin-responsive vesicle compartment of glucose transporter isoform 4-deficient cardiomyocytes.
- Evidence for mitochondrial thioesterase 1 as a peroxisome proliferator-activated receptor-alpha-regulated gene in cardiac and skeletal muscle.
- Impaired cardiac efficiency and increased fatty acid oxidation in insulin-resistant ob/ob mouse hearts.
- Insulin signaling in heart muscle: lessons from genetically engineered mouse models.
- Optical mapping of propagation changes induced by elevated extracellular potassium ion concentration in genetically altered mouse hearts.
- Erythropoietin prevents the acute myocardial inflammatory response induced by ischemia/reperfusion via induction of AP-1.
- The intrinsic circadian clock within the cardiomyocyte.
- Mouse and human resistins impair glucose transport in primary mouse cardiomyocytes, and oligomerization is required for this biological action.
- Akt1 in the cardiovascular system: friend or foe?
- Myocardial insulin resistance and cardiac complications of diabetes.
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