Dr. Maria L. Wei M.D., PH.D.
Dermatologist
4150 Clement St Va 190 4150 Clement Street San Francisco CA, 94121About
Dr. Maria L. Wei is a dermatologist who specializes in melanoma and disorders of pigmentation (acquired and inherited). She is Director of the UCSF Melanoma Surveillance clinic that sees individuals with ...
Education and Training
Duke University School of Medicine 1992
Board Certification
DermatologyAmerican Board of DermatologyABD
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Genetic engineering of Serratia marcescens with bacterial hemoglobin gene: effects on growth, oxygen utilization, and cell size.
- Metabolic engineering of Serratia marcescens with the bacterial hemoglobin gene: alterations in fermentation pathways.
- The splicing determinants of a regulated exon in the axonal MAP tau reside within the exon and in its upstream intron.
- Incidence of transitional cell carcinoma and arsenic in drinking water: a follow-up study of 8,102 residents in an arseniasis-endemic area in northeastern Taiwan.
- Melanosome morphologies in murine models of hermansky-pudlak syndrome reflect blocks in organelle development.
- Transport and expression of class I MHC glycoproteins in an antigen-processing mutant cell line.
- Characterization of melanosomes in murine Hermansky-Pudlak syndrome: mechanisms of hypopigmentation.
- HLA-A2 molecules in an antigen-processing mutant cell contain signal sequence-derived peptides.
- The origins of the thenar and hypothenar muscles.
- Hermansky-Pudlak syndrome: a disease of protein trafficking and organelle function.
- Hermansky-Pudlak HPS1/pale ear gene regulates epidermal and dermal melanocyte development.
- Genetic engineering to contain the Vitreoscilla hemoglobin gene enhances degradation of benzoic acid by Xanthomonas maltophilia.
- Multidrug resistance decreases with mutations of melanosomal regulatory genes.
- The MADS box transcription factor MEF2C regulates melanocyte development and is a direct transcriptional target and partner of SOX10.
- Targeting protein-trafficking pathways alters melanoma treatment sensitivity.
Treatments
- Melanoma
- Eczema
- Dermatitis
- Moles
- Warts
- Actinic Keratosis
- Skin Cancer
- Birthmark
- Contact Dermatitis
- Vitiligo
- Seborrheic Keratosis
Fellowships
- UCSF Medical Center, Biochemistry and Biophysics 2002
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