Dr. Nitin Karandikar is a pathologist practicing in Dallas, TX. Dr. Karandikar is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Karandikar can help your primary care doctor make a diagnosis about your medical condition. Dr. Karandikar may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Education and Training
B.J. Medical College, Pune, Maharashtra, India 1990
PathologyAmerican Board of PathologyABP- Hematology Pathology
- The role of CTLA-4 in induction and maintenance of peripheral T cell tolerance.
- High prevalence of autoreactive, neuroantigen-specific CD8+ T cells in multiple sclerosis revealed by novel flow cytometric assay.
- CTLA-4 regulates expansion and differentiation of Th1 cells following induction
- Haematogones in the peripheral blood of adults: a four-colour flow cytometry study of 102 patients.
- Therapeutic induction of regulatory, cytotoxic CD8+ T cells in multiple sclerosis.
- Glatiramer acetate (GA) therapy induces a focused, oligoclonal CD8+ T-cell repertoire in multiple sclerosis.
- Transient regulatory T-cells: a state attained by all activated human T-cells.
- Immunophenotypic differentiation between neoplastic plasma cells in mature B-cell lymphoma vs plasma cell myeloma.
- Differential dysfunction in dendritic cell subsets during chronic HCV infection.
- Clinical responders to antiviral therapy of chronic HCV infection show elevated antiviral CD4+ and CD8+ T-cell responses.
- In vitro methotrexate as a practical approach to selective allodepletion.
- Human regulatory T cells: a unique, stable thymic subset or a reversible peripheral state of differentiation?
- Attack on the clones? Human FOXP3 detection by PCH101, 236A/E7, 206D, and 259D reveals 259D as the outlier with lower sensitivity.
- Decrease in the numbers of dendritic cells and CD4+ T cells in cerebral perivascular spaces due to natalizumab.
- Immunophenotypic studies of monoclonal gammopathy of undetermined significance.
- Bright CD38 expression is an indicator of MYC rearrangement.
- The oligodendrocyte-specific G protein-coupled receptor GPR17 is a cell-intrinsic timer of myelination.
- Significant CD5 expression on normal stage 3 hematogones and mature B Lymphocytes in bone marrow.
- The mechanism of action of glatiramer acetate treatment in multiple sclerosis.
- Immune regulatory CNS-reactive CD8+T cells in experimental autoimmune encephalomyelitis.
- Simple karyotype and bcl-6 expression predict a diagnosis of Burkitt lymphoma and better survival in IG-MYC rearranged high-grade B-cell lymphomas.
- Notch1 in primary effusion lymphoma: a clinicopathological study.
- Acute myeloid leukemia with inv(16) with CBFB-MYH11, 3'CBFB deletion, variant t(9;22) with BCR-ABL1, and del(7)(q22q32) in a pediatric patient: case report and literature review.
- Elucidation of seventeen human peripheral blood B-cell subsets and quantification of the tetanus response using a density-based method for the automated identification of cell populations in multidimensional flow cytometry data.
- Neuroantigen-specific CD8+ regulatory T-cell function is deficient during acute exacerbation of multiple sclerosis.
- Clonal composition of neuroantigen-specific CD8+ and CD4+ T-cells in multiple sclerosis.
- Viral interactions with B-cells contribute to increased regulatory T-cells during chronic HCV infection.
- Distinct immunophenotype of early T-cell progenitors in T lymphoblastic leukemia/lymphoma may predict FMS-like tyrosine kinase 3 mutations.
- Modulation of immune function occurs within hours of therapy initiation for multiple sclerosis.
- The disease-ameliorating function of autoregulatory CD8 T cells is mediated by
- CD8(+) T Cells Are Required For Glatiramer Acetate Therapy in Autoimmune Demyelinating Disease.
- Disease exacerbation of multiple sclerosis is characterized by loss of terminally differentiated autoregulatory CD8+ T cells.
- Immune regulation of multiple sclerosis by CD8+ T cells.
- Neuroantigen-specific autoregulatory CD8+ T cells inhibit autoimmune demyelination through modulation of dendritic cell function.
- Acute Megakaryoblastic Leukemia Associated with Trisomy 21 Demonstrates a
- Acute megakaryoblastic leukemia associated with trisomy 21 demonstrates a distinct immunophenotype.
- Multiparameter Flow Cytometric Assays to Quantify Effector and Regulatory T-Cell Function in Multiple Sclerosis.
- Autoregulatory CD8 T cells depend on cognate antigen recognition and CD4/CD8 myelin determinants.
- CD8(+) T-Cells as Immune Regulators of Multiple Sclerosis.
- A method for histopathological study of the multifocal nature of spinal cord lesions in murine experimental autoimmune encephalomyelitis.
- Leukemic Transdifferentiation of Follicular Lymphoma Into an Acute Histiocytic Leukemia in a 52-Year-Old Caucasian Woman.
- Induction of regulatory T-cells from memory T-cells is perturbed during acute exacerbation of multiple sclerosis.
- Suppression of autoimmune demyelinating disease by preferential stimulation of CNS-specific CD8 T cells using Listeria-encoded neuroantigen.
- Using Focused Laboratory Management and Quality Improvement Projects to Enhance Resident Training and Foster Scholarship.
- CTLA-4: a negative regulator of autoimmune disease.
- Blockade of CD28/B7-1 interaction prevents epitope spreading and clinical relapses of murine EAE.
- Degenerate antigen recognition by CD4+ effector T cells in experimental autoimmune encephalomyelitis.
- Tissue-specific up-regulation of B7-1 expression and function during the course
- Targeting the B7/CD28:CTLA-4 costimulatory system in CNS autoimmune disease.
- Univ Tx Southwestern Med Sch, Anatomic And Clinical Pathology
Dr. Nitin J Karandikar MD PHD's Practice location
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IOWA CITY, IA 52242Get Direction
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