Dr. Gregory J Gores M.D.
Gastroenterologist | Gastroenterology
200 1st St SW Rochester MN, 55905About
Dr. Gregory Gores is a gastroenterologist practicing in Rochester, MN. Dr. Gores specializes in the digestive system and its diseases that affect the gastrointestinal tract, which include organs from the mouth to the anus as well as liver disorders. Gastroenterology includes conditions such as hepatitis, peptic ulcer disease, colitis, nutritional problems and irritable bowel syndrome. Dr. Gores performs colonoscopy and endoscopy procedures and provides accurate and thorough care for patients suffering from digestive issues.
Education and Training
Univ Of Nd Sch Of Med- Grand Forks Nd 1980
University of North Dakota School of Medicine and Health Sciences 1980
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Mechanisms of hepatotoxicity.
- COX-2 inhibits Fas-mediated apoptosis in cholangiocarcinoma cells.
- Bile acids induce cyclooxygenase-2 expression via the epidermal growth factor receptor in a human cholangiocarcinoma cell line.
- Nitric oxide inhibits apoptosis downstream of cytochrome C release by nitrosylating caspase 9.
- Ursodeoxycholic acid cytoprotection: dancing with death receptors and survival pathways.
- Increased expression of cyclooxygenase-2 in human pancreatic neoplasms and potential for chemoprevention by cyclooxygenase inhibitors.
- In primary sclerosing cholangitis, gallbladder polyps are frequently malignant.
- Yes, hepatocellular cancer does occur in primary biliary cirrhosis.
- Death receptor-mediated apoptosis and the liver.
- Tumor necrosis factor-alpha-associated lysosomal permeabilization is cathepsin B dependent.
- p16INK4a promoter mutations are frequent in primary sclerosing cholangitis (PSC) and PSC-associated cholangiocarcinoma.
- Fas enhances fibrogenesis in the bile duct ligated mouse: a link between apoptosis and fibrosis.
- Cholestasis increases tumor necrosis factor-related apoptotis-inducing ligand (TRAIL)-R2/DR5 expression and sensitizes the liver to TRAIL-mediated cytotoxicity.
- Bile acids stimulate cFLIP phosphorylation enhancing TRAIL-mediated apoptosis.
- Bile acids inhibit Mcl-1 protein turnover via an epidermal growth factor receptor/Raf-1-dependent mechanism.
Treatments
- Liver Cancer
- Bile Duct Cancer
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