Andreas Sebastian Barth M.D.
Cardiologist | Cardiovascular Disease
4940 EASTERN AVE BALTIMORE MD, 21224About
Dr. Andreas Barth is a cardiologist practicing in BALTIMORE, MD. Dr. Barth specializes in diagnosing, monitoring, and treating diseases or conditions of the heart and blood vessels and the cardiovascular system. These conditions include heart attacks, heart murmurs, coronary heart disease, and hypertension. Dr. Barth also practices preventative medicine, helping patients maintain a heart-healthy life.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Global gene expression in human myocardium-oligonucleotide microarray analysis of regional diversity and transcriptional regulation in heart failure.
- Novel KChIP2 isoforms increase functional diversity of transient outward potassium currents.
- Reprogramming of the human atrial transcriptome in permanent atrial fibrillation: expression of a ventricular-like genomic signature.
- Functional profiling of human atrial and ventricular gene expression.
- MAPK= mitogen-activated protein KChIP2? Unraveling signaling pathways controlling cardiac Ito expression.
- Interventional embolization of a giant pulmonary arteriovenous malformation with right-left-shunt associated with hereditary hemorrhagic telangiectasia.
- Gene transfer of a synthetic pacemaker channel into the heart: a novel strategy for biological pacing.
- Identification of a common gene expression signature in dilated cardiomyopathy across independent microarray studies.
- Lentiviral vector-mediated expression of GFP or Kir2.1 alters the electrophysiology of neonatal rat ventricular myocytes without inducing cytotoxicity.
- CAPON modulates cardiac repolarization via neuronal nitric oxide synthase signaling in the heart.
- Genomic analysis reveals poor separation of human cardiomyopathies of ischemic and nonischemic etiologies.
- Key pathways associated with heart failure development revealed by gene networks correlated with cardiac remodeling.
- Cardiac metabolism and arrhythmias.
- Isolation and expansion of functionally-competent cardiac progenitor cells directly from heart biopsies.
- Sudden cardio arrest: oxidative stress irritates the heart.
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