Dr. Jeffrey David Klopfenstein M.D.
Neurosurgeon
719 N William Kumpf Blvd Suite 100 Peoria IL, 61605About
Dr. Jeffrey Klopfenstein practices Neurological Surgery in Peoria, IL. As a Neurological Surgeon, Dr. Klopfenstein prevents, diagnoses, evaluates, and treats disorders of the autonomic, peripheral, and central nervous systems. Neurological Surgeons are trained to treat such disorders as spinal canal stenosis, herniated discs, tumors, fractures, and spinal deformities, among many others.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Scalp burns from halo pins following magnetic resonance imaging. Case illustration.
- MMP-2 downregulation mediates differential regulation of cell death via ErbB-2 in glioma xenografts.
- Human umbilical cord blood stem cells show PDGF-D-dependent glioma cell tropism in vitro and in vivo.
- Regulation of glioblastoma progression by cord blood stem cells is mediated by downregulation of cyclin D1.
- α3β1 integrin promotes radiation-induced migration of meningioma cells.
- Anterior communicating artery aneurysm rupture after intravenous thrombolysis for acute middle cerebral artery thromboembolism: case report.
- MMP-9 silencing regulates hTERT expression via β1 integrin-mediated FAK signaling and induces senescence in glioma xenograft cells.
- Chk2-mediated G2/M cell cycle arrest maintains radiation resistance in malignant meningioma cells.
- Metabolic remodeling precedes mitochondrial outer membrane permeabilization in human glioma xenograft cells.
- SPARC overexpression inhibits cell proliferation in neuroblastoma and is partly mediated by tumor suppressor protein PTEN and AKT.
- Incidence of neurosurgical wrong-site surgery before and after implementation of the universal protocol.
- Temporal regulation of apoptotic and anti-apoptotic molecules after middle cerebral artery occlusion followed by reperfusion.
- Incidence and ramifications of the oculocardiac reflex during the orbitozygomatic approach: a prospective assessment.
- Stem cell treatment after cerebral ischemia regulates the gene expression of apoptotic molecules.
- Post-transcriptional inactivation of matrix metalloproteinase-12 after focal cerebral ischemia attenuates brain damage.
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