Dr. Jeffrey Michael Craig MD
Neurologist | Neurology
505 Parnassus Ave # M-1202 San Francisco CA, 94143About
Dr. Jeffrey Craig is a distinguished Neurologist in San Francisco, CA. Dr. Craig specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Craig employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Craig is committed to staying abreast of the latest developments in neurological research and therapies.
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN- Vascular Neurology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Centromeric chromatin pliability and memory at a human neocentromere.
- A rapid method of genomic array analysis of scaffold/matrix attachment regions (S/MARs) identifies a 2.5-Mb region of enhanced scaffold/matrix attachment at a human neocentromere.
- Analysis of mammalian proteins involved in chromatin modification reveals new metaphase centromeric proteins and distinct chromosomal distribution patterns.
- Transcription within a functional human centromere.
- QTc prolongation: possible association with risperidone and/or haloperidol.
- Effects of scaffold/matrix alteration on centromeric function and gene expression.
- Heterochromatin--many flavours, common themes.
- Brahma links the SWI/SNF chromatin-remodeling complex with MeCP2-dependent transcriptional silencing.
- Kiss and break up--a safe passage to anaphase in mitosis and meiosis.
- Harry Potter and the recessive allele.
- Permissive transcriptional activity at the centromere through pockets of DNA hypomethylation.
- SmcHD1, containing a structural-maintenance-of-chromosomes hinge domain, has a critical role in X inactivation.
- Prospects for epigenetic epidemiology.
- Placenta-specific methylation of the vitamin D 24-hydroxylase gene: implications for feedback autoregulation of active vitamin D levels at the fetomaternal interface.
- DNA methylation-mediated down-regulation of DNA methyltransferase-1 (DNMT1) is coincident with, but not essential for, global hypomethylation in human placenta.
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