Michael Alan Laflamme
Pathologist | Anatomic Pathology & Clinical Pathology
University Of Washington Medical Ctr 1959 Ne Pacific St Seattle WA, 98195About
Dr. Michael Laflamme is a pathologist practicing in Seattle, WA. Dr. Laflamme is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Laflamme can help your primary care doctor make a diagnosis about your medical condition. Dr. Laflamme may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Human embryonic stem cells and cardiac repair.
- Cardiogenic differentiation and transdifferentiation of progenitor cells.
- Local control of excitation-contraction coupling in human embryonic stem cell-derived cardiomyocytes.
- Cardiac applications for human pluripotent stem cells.
- Neuregulin/ErbB signaling regulates cardiac subtype specification in differentiating human embryonic stem cells.
- Efficient generation and cryopreservation of cardiomyocytes derived from human embryonic stem cells.
- Methods for the derivation and use of cardiomyocytes from human pluripotent stem cells.
- Human ES-cell-derived cardiomyocytes electrically couple and suppress arrhythmias in injured hearts.
- Targeted genomic integration of a selectable floxed dual fluorescence reporter in human embryonic stem cells.
- Structural and functional maturation of cardiomyocytes derived from human pluripotent stem cells.
- Human embryonic stem cell-derived cardiomyocytes migrate in response to gradients of fibronectin and Wnt5a.
- Biowire: a platform for maturation of human pluripotent stem cell-derived cardiomyocytes.
- Electrical Integration of Human Embryonic Stem Cell-Derived Cardiomyocytes in a
- Myocardial deletion of transcription factor CHF1/Hey2 results in altered myocyte action potential and mild conduction system expansion but does not alter conduction system function or promote spontaneous arrhythmias.
- Cell-based delivery of dATP via gap junctions enhances cardiac contractility.
Fellowships
- UW - Cardiovascular Pathology Fellowship
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Nearby Providers
- Dr. Craig Jonov DMD, MD509 Olive Way Seattle WA 98101
- Dr. Christopher Gault M.D.1100 9th Ave Seattle WA 98101
- Dr. Veronica Thoroughgood M.D.1280 116th Ave Ne Bellevue WA 98004
- Dr. S Nicholas Agoff MD1100 9th Ave Seattle WA 98101
- Dr. Brian A Folz M.D.1001 Sw Klickitat Way Seattle WA 98134
- Dr. Sandra Pauline Ewaskow MD1280 116th Ave Ne Bellevue WA 98004