Dr. William Ted Brown MD, PHD
Geneticist | Clinical Genetics (M.D.)
1050 Forest Hill Rd Nys Institute For Ba Staten Island NY, 10314About
Dr. William Brown practices Genetic Medicine in Staten Island, NY. As a geneticist, Dr. Brown performs experiments and analyzes data to interpret the inheritance of different traits in patients. A geneticist evaluates, diagnoses, and manages patients with hereditary conditions or congenital malformations, genetic risk calculations, and mutation analysis. Dr. Brown carries out studies, tests, and counsels patients with genetic diseases.
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Expert Publications
Data provided by the National Library of Medicine- Long-term changes in calbindin D(28K) immunoreactivity in the rat hippocampus after cardiac arrest.
- Human chromatid ultrastructure: new observations with scanning and transmission
- Increased oxidative stress and decreased activities of Ca(2+)/Mg(2+)-ATPase and
- Fragile X premutation tremor/ataxia syndrome: molecular, clinical, and neuroimaging correlates.
- Recurrent de novo point mutations in lamin A cause Hutchinson-Gilford progeria syndrome.
- Hyaluronan is not elevated in urine or serum in Hutchinson-Gilford Progeria Syndrome.
- Alteration in amino-glycerophospholipids levels in the plasma of children with autism: a potential biochemical diagnostic marker.
- Prospects for gene therapy in the fragile X syndrome.
- Neuropsychiatric symptoms of fragile X syndrome: pathophysiology and pharmacotherapy.
- Oxidative stress in autism: increased lipid peroxidation and reduced serum levels of ceruloplasmin and transferrin--the antioxidant proteins.
- Age-associated memory changes in adults with williams syndrome.
- Prenatal diagnostic testing for infantile and late-infantile neuronal ceroid lipofusinoses (NCL) using allele specific primer extension (ASPE).
- Decreased GABA(A) receptor expression in the seizure-prone fragile X mouse.
- Novel progerin-interactive partner proteins hnRNP E1, EGF, Mel 18, and UBC9 interact with lamin A/C.
- Genotype-phenotype analyses of classic neuronal ceroid lipofuscinosis (NCLs): genetic predictions from clinical and pathological findings.
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