Dr. Leif William Ellisen MD PHD
Oncologist | Medical Oncology
55 Fruit St Grj 904 Boston MA, 02114About
Dr. Leif Ellisen is an oncologist practicing in Boston, MA. Dr. Ellisen specializes in the care and treatment of patients with cancer. As an oncologist, Dr. Ellisen manages and oversees the treatment of a cancer patient after he or she has been diagnosed with the disease. Oncologists will care for their patients throughout the course of the disease. Types of oncologists include medical oncologists, surgical oncologists, radiation oncologists, gynecologic oncologists, pediatric oncologists and hematologist oncologists.
Education and Training
Stanford Univ Sch Of Med- Stanford Ca 1992
Stanford University School of Medicine 1992
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Induction of GADD45 and JNK/SAPK-dependent apoptosis following inducible expression of BRCA1.
- A functional interaction with CBP contributes to transcriptional activation by the Wilms tumor suppressor WT1.
- Regulation of gene expression by WT1 in development and tumorigenesis.
- REDD1, a developmentally regulated transcriptional target of p63 and p53, links p63 to regulation of reactive oxygen species.
- Regulation of mTOR and cell growth in response to energy stress by REDD1.
- Growth control under stress: mTOR regulation through the REDD1-TSC pathway.
- p63 mediates survival in squamous cell carcinoma by suppression of p73-dependent apoptosis.
- p63 and p73: life and death in squamous cell carcinoma.
- p63 regulates an adhesion programme and cell survival in epithelial cells.
- Tumor-specific p73 up-regulation mediates p63 dependence in squamous cell carcinoma.
- Dexamethasone represses signaling through the mammalian target of rapamycin in muscle cells by enhancing expression of REDD1.
- p63 and p73 in human cancer: defining the network.
- The p63/p73 network mediates chemosensitivity to cisplatin in a biologically defined subset of primary breast cancers.
- Hypoxia regulates TSC1/2-mTOR signaling and tumor suppression through REDD1-mediated 14-3-3 shuttling.
- Reciprocal intraepithelial interactions between TP63 and hedgehog signaling regulate quiescence and activation of progenitor elaboration by mammary stem cells.
Treatments
- Breast Cancer
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