Dr. James R Davie M.D., PH.D.
Pathologist | Anatomic Pathology & Clinical Pathology
15936 Meadow Vista Pl Dallas TX, 75248About
Dr. James Davie is a pathologist practicing in Dallas, TX. Dr. Davie is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Davie can help your primary care doctor make a diagnosis about your medical condition. Dr. Davie may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Board Certification
PathologyAmerican Board of PathologyABP- Dermatopathology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Purification and characterization of chicken erythrocyte histone deacetylase 1.
- Increased Ser-10 phosphorylation of histone H3 in mitogen-stimulated and oncogene-transformed mouse fibroblasts.
- Role of covalent modifications of histones in regulating gene expression.
- Nuclear matrix proteins associated with DNA in situ in hormone-dependent and hormone-independent human breast cancer cell lines.
- Tamoxifen-bound estrogen receptor (ER) strongly interacts with the nuclear matrix
- The human factors YY1 and LSF repress the human immunodeficiency virus type 1 long terminal repeat via recruitment of histone deacetylase 1.
- Rapid induction of histone hyperacetylation and cellular differentiation in human breast tumor cell lines following degradation of histone deacetylase-1.
- Expression of E1 component of human branched-chain alpha-keto acid dehydrogenase complex in Escherichia coli by cotransformation with chaperonins GroEL and GroES.
- Production of recombinant mammalian holo-E2 and E3 and reconstitution of functional branched-chain alpha-keto acid dehydrogenase complex with recombinant E1.
- CUG-initiated FGF-2 induces chromatin compaction in cultured cardiac myocytes and in vitro.
- Altered profiles in nuclear matrix proteins associated with DNA in situ during progression of breast cancer cells.
- Control of chromatin remodeling.
- An essential role for Mad homology domain 1 in the association of Smad3 with histone deacetylase activity*.
- Signal transduction pathways and chromatin structure in cancer cells.
- Dynamically acetylated histone association with transcriptionally active and competent genes in the avian adult beta-globin gene domain.
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