Dr. Thomas C Friedrich MD
Orthopedist
701 E Roosevelt Blvd Bldg 600 Monroe NC, 28112About
Dr. Thomas Friedrich is an orthopaedic surgeon practicing in Monroe, NC. Dr. Friedrich specializes in the diagnosis, treatment and rehabilitation of injuries, diseases and disorders of the bodys musculoskeletal system. As an orthopaedic surgeon, Dr. Friedrich tends to bones, ligaments, muscles, joints, nerves and tendons. Orthopaedic surgeons can specialize in certain areas like the hand, spine, hip, foot and ankle, shoulder and elbow or the knee.
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Escape in one of two cytotoxic T-lymphocyte epitopes bound by a high-frequency major histocompatibility complex class I molecule, Mamu-A*02: a paradigm for virus evolution and persistence?
- Consequences of cytotoxic T-lymphocyte escape: common escape mutations in simian immunodeficiency virus are poorly recognized in naive hosts.
- The role of the SIV model in AIDS vaccine research.
- Vaccine-induced cellular immune responses reduce plasma viral concentrations after repeated low-dose challenge with pathogenic simian immunodeficiency virus SIVmac239.
- Repeated intravaginal inoculation with cell-associated simian immunodeficiency virus results in persistent infection of nonhuman primates.
- Simian immunodeficiency virus SIVmac239 infection of major histocompatibility complex-identical cynomolgus macaques from Mauritius.
- Not all cytokine-producing CD8+ T cells suppress simian immunodeficiency virus replication.
- The antiviral efficacy of simian immunodeficiency virus-specific CD8+ T cells is unrelated to epitope specificity and is abrogated by viral escape.
- Subdominant CD8+ T-cell responses are involved in durable control of AIDS virus replication.
- Pol-specific CD8+ T cells recognize simian immunodeficiency virus-infected cells prior to Nef-mediated major histocompatibility complex class I downregulation.
- AIDS virus specific CD8+ T lymphocytes against an immunodominant cryptic epitope select for viral escape.
- Patterns of CD8+ immunodominance may influence the ability of Mamu-B*08-positive macaques to naturally control simian immunodeficiency virus SIVmac239 replication.
- Genital ulcers facilitate rapid viral entry and dissemination following intravaginal inoculation with cell-associated simian immunodeficiency virus SIVmac239.
- Comprehensive immunological evaluation reveals surprisingly few differences between elite controller and progressor Mamu-B*17-positive simian immunodeficiency virus-infected rhesus macaques.
- Differential antigen presentation kinetics of CD8+ T-cell epitopes derived from the same viral protein.
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Nearby Providers
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