Dr. John Murray Greally M.B., PH.D
Geneticist | Clinical Genetics (M.D.)
3415 Bainbridge Ave Cham Bronx NY, 10467About
Dr. John Greally practices Genetic Medicine in Bronx, NY. As a geneticist, Dr. Greally performs experiments and analyzes data to interpret the inheritance of different traits in patients. A geneticist evaluates, diagnoses, and manages patients with hereditary conditions or congenital malformations, genetic risk calculations, and mutation analysis. Dr. Greally carries out studies, tests, and counsels patients with genetic diseases.
Education and Training
Tbilisi National University 'Gaenati' 1988
Board Certification
Medical GeneticsAmerican Board of Medical GeneticsABMG
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Report of the fourth international workshop on human chromosome 15 mapping 1997.
- Genomic imprinting and chromatin insulation in Beckwith-Wiedemann syndrome.
- Conserved characteristics of heterochromatin-forming DNA at the 15q11-q13 imprinting center.
- Beta-globin YAC transgenes exhibit uniform expression levels but position effect variegation in mice.
- Position effects are influenced by the orientation of a transgene with respect to flanking chromatin.
- A triple color FISH technique for mouse chromosome identification.
- Short interspersed transposable elements (SINEs) are excluded from imprinted regions in the human genome.
- Genomic organization of the genes Gtf2ird1, Gtf2i, and Ncf1 at the mouse chromosome 5 region syntenic to the human chromosome 7q11.23 Williams syndrome critical region.
- Retrotransposed genes such as Frat3 in the mouse Chromosome 7C Prader-Willi syndrome region acquire the imprinted status of their insertion site.
- Identification of a candidate regulatory region in the human CD8 gene complex by colocalization of DNase I hypersensitive sites and matrix attachment regions which bind SATB1 and GATA-3.
- Mammalian linker-histone subtypes differentially affect gene expression in vivo.
- A stain upon the silence: genes escaping X inactivation.
- Identification of four highly conserved genes between breakpoint hotspots BP1 and BP2 of the Prader-Willi/Angelman syndromes deletion region that have undergone evolutionary transposition mediated by flanking duplicons.
- Epigenomics: beyond CpG islands.
- Genetic mapping of putative Chrna7 and Luzp2 neuronal transcriptional enhancers due to impact of a transgene-insertion and 6.8 Mb deletion in a mouse model of Prader-Willi and Angelman syndromes.
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