Arturo Martin Valverde MD
Cardiac Electrophysiologist | Clinical Cardiac Electrophysiology
1400 Bellinger St Eau Claire WI, 54703About
Dr. Arturo Valverde is a Cardiac Electrophysicist practicing in Eau Claire, WI. Dr. Valverde performs testing procedures on patients hearts electrical system in order to assess timing and abnormalities associated with electrical activities of the heart. Adult Cardiac Electrophysiologists are trained to treat irregular heartbeats, and carry out electrophysiology studies or ablations.
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Expert Publications
Data provided by the National Library of Medicine- Activated Ha-ras induces apoptosis by association with phosphorylated Bcl-2 in a mitogen-activated protein kinase-independent manner.
- Insulin signaling in insulin receptor substrate (IRS)-1-deficient brown adipocytes: requirement of IRS-1 for lipid synthesis.
- Okadaic acid inhibits insulin-induced glucose transport in fetal brown adipocytes in an Akt-independent and protein kinase C zeta-dependent manner.
- Epidermal growth factor impairs the cytochrome C/caspase-3 apoptotic pathway induced by transforming growth factor beta in rat fetal hepatocytes via a phosphoinositide 3-kinase-dependent pathway.
- Akt mediates insulin rescue from apoptosis in brown adipocytes: effect of ceramide.
- Insulin produces myogenesis in C2C12 myoblasts by induction of NF-kappaB and downregulation of AP-1 activities.
- Association of insulin receptor substrate 1 (IRS-1) y895 with Grb-2 mediates the insulin signaling involved in IRS-1-deficient brown adipocyte mitogenesis.
- Brown adipose tissue-specific insulin receptor knockout shows diabetic phenotype without insulin resistance.
- Phosphoenolpyruvate carboxykinase and glucose-6-phosphate dehydrogenase expression in fetal hepatocyte primary cultures under proliferative conditions.
- The brown adipose cell: a model for understanding the molecular mechanisms of insulin resistance.
- Differential mitogenic signaling in insulin receptor-deficient fetal pancreatic beta-cells.
- Hormonal regulation of malic enzyme and glucose-6-phosphate-dehydrogenase expression in fetal brown-adipocyte primary cultures under non-proliferative conditions.
- Proliferation of fetal brown adipocyte primary cultures: relationship with the genetic expression of glucose 6 phosphate dehydrogenase.
- Protein tyrosine phosphatase 1B modulates GSK3β/Nrf2 and IGFIR signaling pathways in acetaminophen-induced hepatotoxicity.
- Insulin directly stimulates VEGF-A production in the glomerular podocyte.
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