Dr. Kenneth R Feingold MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
Metabolism 111f, Va Medical Center 4150 Clement St San Francisco CA, 94121About
Dr. Kenneth Feingold practices Endocrinology in San Francisco, CA. Dr. Feingold specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Feingold examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Suny-Hlth Sci Ctr At Syracuse, Coll of Med, Syracuse Ny 1974
Upstate Medical University/ College of Health Professions 1974
State University of New York Upstate Medical University 1974
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Induction of selected lipid metabolic enzymes and differentiation-linked structural proteins by air exposure in fetal rat skin explants.
- Cloning of murine translation initiation factor 6 and functional analysis of the homologous sequence YPR016c in Saccharomyces cerevisiae.
- Effect of oxidized lipids in the diet on oxidized lipid levels in postprandial serum chylomicrons of diabetic patients.
- Regulation of glycosphingolipid metabolism in liver during the acute phase response.
- In vivo regulation of plasma platelet-activating factor acetylhydrolase during the acute phase response.
- Cutaneous barrier repair and pathophysiology following barrier disruption in IL-1 and TNF type I receptor deficient mice.
- Epidermal pathogenesis of inflammatory dermatoses.
- Down-regulation of liver and heart specific fatty acid binding proteins by endotoxin and cytokines in vivo.
- Fetal epidermal differentiation and barrier development In vivo is accelerated by nuclear hormone receptor activators.
- Plasma platelet-activating factor acetylhydrolase activity in human immunodeficiency virus infection and the acquired immunodeficiency syndrome.
- Coordinate assembly of lipids and enzyme proteins into epidermal lamellar bodies.
- Stress alters cutaneous permeability barrier homeostasis.
- Leptin-deficient (ob/ob) mice are protected from T cell-mediated hepatotoxicity: role of tumor necrosis factor alpha and IL-18.
- Upregulation of uncoupling protein 2 mRNA in genetic obesity: lack of an essential role for leptin, hyperphagia, increased tissue lipid content, and TNF-alpha.
- Oxysterols induce differentiation in human keratinocytes and increase Ap-1-dependent involucrin transcription.
Treatments
- Diabetes
- Osteoporosis
- Type 2 Diabetes
- Hyperthyroidism
- Hypothyroidism
- Graves' Disease
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