Dr. Marie Eloise Egan MD
Pulmonologist (Pediatric) | Pediatric Pulmonology
20 York St Ynhh (children's) We New Haven CT, 06504Rating
5/5
About
Dr. Marie Egan practices Pediatric Pulmonology in New Haven, CT. Dr. Egan treats children who have breathing problems, or a problem with his or her lungs. Pediatric pulmonologists often treat children with chronic cough, difficulty breathing, recurring pneumonia, asthma, cystic fibrosis, apnea, chronic lung disease in premature infants, noisy breathing, and conditions that require special equipment to monitor and/or help with breathing at home.
Board Certification
PediatricsAmerican Board of PediatricsABP- Pediatric Pulmonology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- CFTR-associated ATP transport and release.
- DeltaF508 mutation results in impaired gastric acid secretion.
- Rectal potential difference and the functional expression of CFTR in the gastrointestinal epithelia in cystic fibrosis mouse models.
- Macrophages directly contribute to the exaggerated inflammatory response in cystic fibrosis transmembrane conductance regulator-/- mice.
- Pulmonary squamous cell carcinoma associated with repaired congenital tracheoesophageal fistula and esophageal atresia.
- Mouse cystic fibrosis transmembrane conductance regulator forms cAMP-PKA-regulated apical chloride channels in cortical collecting duct.
- Calcium-modulated chloride pathways contribute to chloride flux in murine cystic fibrosis-affected macrophages.
- Myosin Ia is required for CFTR brush border membrane trafficking and ion transport in the mouse small intestine.
- Surface modified poly(β amino ester)-containing nanoparticles for plasmid DNA delivery.
- Baby bottle steam sterilizers disinfect home nebulizers inoculated with bacterial respiratory pathogens.
- Reduced caveolin-1 promotes hyperinflammation due to abnormal heme oxygenase-1 localization in lipopolysaccharide-challenged macrophages with dysfunctional cystic fibrosis transmembrane conductance regulator.
- Pharmacological modulation of the AKT/microRNA-199a-5p/CAV1 pathway ameliorates cystic fibrosis lung hyper-inflammation.
- Nanoparticles that deliver triplex-forming peptide nucleic acid molecules correct F508del CFTR in airway epithelium.
- Restoration of cytoskeletal and membrane tethering defects but not defects in membrane trafficking in the intestinal brush border of mice lacking both myosin Ia and myosin VI.
- Increased susceptibility of Cftr-/- mice to LPS-induced lung remodeling.
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300 SEASIDE AVENUE MILFORD CT 6460