Dr. Robert Craig Castellino MD
Hematologist (Pediatric) | Pediatric Hematology-Oncology
14015 Clifton Rd Atlanta GA, 30322About
Dr. Robert Castellino is a pediatric hematologist practicing in Atlanta, GA. Dr. Castellino specializes in treating children that have a blood disease or cancer. Such blood diseases include disorders of red blood cells, white blood cells and/or platelets. The types of cancers that Dr. Castellino treats include leukemias, lymphomas and certain tumors. Dr. Castellino can also treat bleeding disorders in children. Pediatric hematologists can be found in childrens hospitals, community hospitals, university medical centers and more.
Board Certification
PediatricsAmerican Board of PediatricsABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Medulloblastomas overexpress the p53-inactivating oncogene WIP1/PPM1D.
- HDM2 promotes WIP1-mediated medulloblastoma growth.
- Response to bevacizumab, irinotecan, and temozolomide in children with relapsed medulloblastoma: a multi-institutional experience.
- MEK inhibition increases lapatinib sensitivity via modulation of FOXM1.
- The rationale for targeted therapies in medulloblastoma.
- The WIP1 oncogene promotes progression and invasion of aggressive medulloblastoma variants.
- Insulin-like growth factor-1 receptor signaling increases the invasive potential of human epidermal growth factor receptor 2-overexpressing breast cancer cells via Src-focal adhesion kinase and forkhead box protein M1.
- Mechanisms of environmental chemicals that enable the cancer hallmark of evasion
- WIP1 modulates responsiveness to Sonic Hedgehog signaling in neuronal precursor cells and medulloblastoma.
- in medulloblastoma.
- Clinical responses of patients with diffuse leptomeningeal glioneuronal tumors to chemotherapy.
- Time- and voltage-dependent modulation of a Kv1.4 channel by a beta-subunit (Kv beta 3) cloned from ferret ventricle.
- Bi-stable block by 4-aminopyridine of a transient K+ channel (Kv1.4) cloned from ferret ventricle and expressed in Xenopus oocytes.
- Molecular mechanisms of K+ channel blockade: 4-aminopyridine interaction with a cloned cardiac transient K+ (Kv1.4) channel.
- C-type inactivation controls recovery in a fast inactivating cardiac K+ channel (Kv1.4) expressed in Xenopus oocytes.
Dr. Robert Craig Castellino MD's Practice location
ATLANTA, GA 30342Get Direction
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