Dr. John F Reichard M.D.
Psychiatrist | Psychiatry
63 Monadnock Rd Chestnut Hill MA, 02467About
Dr. John Reichard is a psychiatrist practicing in Chestnut Hill, MA. Dr. Reichard is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. Reichard diagnoses and treats mental illnesses. Dr. Reichard may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. Reichard treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
Education and Training
Harvard Med Sch, Boston Ma 1952
Harvard Medical School 1952
Board Certification
Psychiatry and NeurologyAmerican Board of Psychiatry and NeurologyABPN
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Characterization of 4-hydroxy-2-nonenal metabolism in stellate cell lines derived from normal and cirrhotic rat liver.
- Characterization of multidrug resistance-associated protein 2 in the hepatocellular disposition of 4-hydroxynonenal.
- Hepatic stellate cells lack AP-1 responsiveness to electrophiles and phorbol 12-myristate-13-acetate.
- Arsenite-induced aryl hydrocarbon receptor nuclear translocation results in additive induction of phase I genes and synergistic induction of phase II genes.
- Butylhydroquinone protects cells genetically deficient in glutathione biosynthesis from arsenite-induced apoptosis without significantly changing their prooxidant status.
- Involvement of phosphatidylinositol 3-kinase and extracellular-regulated kinase in hepatic stellate cell antioxidant response and myofibroblastic transdifferentiation.
- Long term low-dose arsenic exposure induces loss of DNA methylation.
- Heme oxygenase-1 induction by NRF2 requires inactivation of the transcriptional repressor BACH1.
- BACH1 is a specific repressor of HMOX1 that is inactivated by arsenite.
- Induction of oxidative stress responses by dioxin and other ligands of the aryl hydrocarbon receptor.
- Genomewide analysis of aryl hydrocarbon receptor binding targets reveals an extensive array of gene clusters that control morphogenetic and developmental programs.
- Effects of arsenic exposure on DNA methylation and epigenetic gene regulation.
- Arsenic toxicology: translating between experimental models and human pathology.
- Toxicokinetic and toxicodynamic considerations when deriving health-based exposure limits for pharmaceuticals.
- Mode-of-action evaluation for the effect of trans fatty acids on low-density lipoprotein cholesterol.
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