Dr. Ernest C Borden MD
Oncologist | Medical Oncology
9500 Euclid Ave Cleveland OH, 44195About
Dr. Ernest Borden is an oncologist practicing in Cleveland, OH. Dr. Borden specializes in the care and treatment of patients with cancer. As an oncologist, Dr. Borden manages and oversees the treatment of a cancer patient after he or she has been diagnosed with the disease. Oncologists will care for their patients throughout the course of the disease. Types of oncologists include medical oncologists, surgical oncologists, radiation oncologists, gynecologic oncologists, pediatric oncologists and hematologist oncologists.
Education and Training
Duke University School of Medicine 1966
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Medical Oncology
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Identification of X-linked inhibitor of apoptosis-associated factor-1 as an
- Resistance to interferons in melanoma cells does not correlate with the expression or activation of signal transducer and activator of transcription 1 (Stat1).
- Interferon stimulated gene 15 constitutively produced by melanoma cells induces e-cadherin expression on human dendritic cells.
- IFN-beta pretreatment sensitizes human melanoma cells to TRAIL/Apo2 ligand-induced apoptosis.
- Malignant melanoma: treatments emerging, but early detection is still key.
- Anticancer activity of sodium stibogluconate in synergy with IFNs.
- Pentamidine is an inhibitor of PRL phosphatases with anticancer activity.
- High-throughput immunoblotting. Ubiquitiin-like protein ISG15 modifies key regulators of signal transduction.
- New and modified interferon alfas: preclinical and clinical data.
- IFN-alpha2b and thalidomide synergistically inhibit tumor-induced angiogenesis.
- Biologic activity of polyethylene glycol12000-interferon-alpha2b compared with
- Suppression of NF-kappa B survival signaling by nitrosylcobalamin sensitizes neoplasms to the anti-tumor effects of Apo2L/TRAIL.
- Novel growth and death related interferon-stimulated genes (ISGs) in melanoma: greater potency of IFN-beta compared with IFN-alpha2.
- Retinoic acid-inducible gene-I is induced by interferon-gamma and regulates the
- Natural soluble interleukin-15Ralpha is generated by cleavage that involves the tumor necrosis factor-alpha-converting enzyme (TACE/ADAM17).
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