Dr. Ralph C Budd M.D.
Rheumatologist | Rheumatology
111 Colchester Ave East Pavillion Level Burlington VT, 05401About
Dr. Ralph Budd is a rheumatologist practicing in Burlington, VT. Dr. Budd specializes in the treatment of musculoskeletal diseases and systematic autoimmune conditions that can affect the bones, muscles or bones. Eventually, if not treated, these illnesses can also impact the skin, eyes, nervous system and internal organs. Dr. Budd treats diseases similar to orthopedists but does not perform surgery. Often times, research is conducted to find potential alternatives for the patients illness.
Education and Training
Jawaharlal Institute of Post Graduate Medicine Research, Pondicherry University 1977
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- A model for the origin of TCR-alphabeta+ CD4-CD8- B220+ cells based on high affinity TCR signals.
- MEK1 activation rescues Jurkat T cells from Fas-induced apoptosis.
- Caspase activation is required for T cell proliferation.
- The caspase-8 inhibitor FLIP promotes activation of NF-kappaB and Erk signaling pathways.
- Activation-induced cell death.
- Liver damage by infiltrating CD8+ T cells is Fas dependent.
- Expression level of c-FLIP versus Fas determines susceptibility to Fas ligand-induced cell death in murine thymoma EL-4 cells.
- Death receptors couple to both cell proliferation and apoptosis.
- Gammadelta T cells promote a Th1 response during coxsackievirus B3 infection in vivo: role of Fas and Fas ligand.
- The caspase 8 inhibitor c-FLIP(L) modulates T-cell receptor-induced proliferation but not activation-induced cell death of lymphocytes.
- High expression of Fas ligand by synovial fluid-derived gamma delta T cells in Lyme arthritis.
- CD2 expression correlates with proliferative capacity of alpha beta + or gamma delta + CD4-CD8- T cells in lpr mice.
- Cellular FLIP long form-transgenic mice manifest a Th2 cytokine bias and enhanced allergic airway inflammation.
- Inhibition of apoptosis and caspase-3 in vascular smooth muscle cells by plasminogen activator inhibitor type-1.
- Accelerated programmed cell death of MRL-lpr/lpr T lymphocytes.
Treatments
- Autoimmune Disorders
- Arthritis
Fellowships
- Dartmouth-Hitchcock Medical Center - Rheumatology Stanford University School of Medicine - Rheumatology
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