Dr. Robert Eugene Stroud DC
Chiropractor
3204 Archdale Rd High Point NC, 27263About
Dr. Robert Stroud is a Chiropractor practicing in High Point, NC. Dr. Stroud specializes in preventing, diagnosing, and treating conditions associated with the neuromusculoskeletal system, while improving each patients functionality and quality of life. Conditions treated include sciatica, neck pain, and arthritis pain, among many others. Dr. Stroud seeks to reduce pain and discomfort through manipulation and adjustment of the spine.
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Expert Publications
Data provided by the National Library of Medicine- Matrix metalloproteinase abundance in human myocardial fibroblasts: effects of sustained pharmacologic matrix metalloproteinase inhibition.
- Direct inhibition of the sodium/hydrogen exchanger after prolonged regional ischemia improves contractility on reperfusion independent of myocardial viability.
- Myocyte contractility with caspase inhibition and simulated hyperkalemic cardioplegic arrest.
- The effects of leukocyte reduction on matrix metalloproteinase release in cardiopulmonary bypass.
- Effects of deletion of the matrix metalloproteinase 9 gene on development of murine thoracic aortic aneurysms.
- Selective targeting of matrix metalloproteinase inhibition in post-infarction myocardial remodeling.
- Alterations in cultured myocardial fibroblast function following the development of left ventricular failure.
- Chronic matrix metalloproteinase inhibition following myocardial infarction in mice: differential effects on short and long-term survival.
- Protein kinase C isoform activation and endothelin-1 mediated defects in myocyte contractility after cardioplegic arrest and reperfusion.
- Differential effects of protein kinase C isoform activation in endothelin-mediated myocyte contractile dysfunction with cardioplegic arrest and reperfusion.
- Temporal disparity in the induction of matrix metalloproteinases and tissue inhibitors of metalloproteinases after thoracic aortic aneurysm formation.
- Interruption of endothelin signaling modifies membrane type 1 matrix metalloproteinase activity during ischemia and reperfusion.
- Relationships between plasma levels of matrix metalloproteinases and
- Altered transforming growth factor-beta signaling in a murine model of thoracic aortic aneurysm.
- Cardiac-restricted overexpression of extracellular matrix metalloproteinase inducer causes myocardial remodeling and dysfunction in aging mice.
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