Dr. Duane Robert Wesemann M.D., PH.D
Internist
75 Francis St Boston MA, 02115Rating
5/5
About
Dr. Duane Wesemann is an internist practicing in Boston, MA. Dr. Wesemann specializes in the medical treatment of adults. Internists can act as a primary physician or a consultant to a primary physician. They manage both common and rare diseases. Dr. Wesemann provides comprehensive care and manages treatment with surgeons as well. Internists establish long-term relationships with their patients and incorporate disease prevention and mental health care into their practice.
Education and Training
University of Alabama School of Medicine 2005
Board Certification
Allergy and ImmunologyAmerican Board of Allergy and ImmunologyABAI
Internal MedicineAmerican Board of Internal MedicineABIM
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Long 5' leaders inhibit removal of a 3' trailer from a precursor tRNA by mammalian tRNA 3' processing endoribonuclease.
- Suppressor of cytokine signaling 1 inhibits cytokine induction of CD40 expression in macrophages.
- STAT-1 alpha and IFN-gamma as modulators of TNF-alpha signaling in macrophages: regulation and functional implications of the TNF receptor 1:STAT-1 alpha complex.
- Molecular regulation of CD40 gene expression in macrophages and microglia.
- Inactivation of wild-type p53 protein function by reactive oxygen and nitrogen species in malignant glioma cells.
- TRADD interacts with STAT1-alpha and influences interferon-gamma signaling.
- Mechanisms promoting translocations in editing and switching peripheral B cells.
- Alternative end-joining catalyzes robust IgH locus deletions and translocations in the combined absence of ligase 4 and Ku70.
- Alternative end-joining catalyzes class switch recombination in the absence of both Ku70 and DNA ligase 4.
- ATM damage response and XLF repair factor are functionally redundant in joining DNA breaks.
- The RNA exosome targets the AID cytidine deaminase to both strands of transcribed duplex DNA substrates.
- Functional redundancy between repair factor XLF and damage response mediator 53BP1 in V(D)J recombination and DNA repair.
- Detection of true IgE-expressing mouse B lineage cells.
- Molecular Mechanisms of IgE Class Switch Recombination.
- Microbes and B cell development.
Treatments
- Immunodeficiency
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300 LONGWOOD AVENUE BOSTON MA 2115