Dr. Barbara Joy Snider MD
Neurologist | Neurology
4488 Forest Park Ave Ground Fl Healthkey Saint Louis MO, 63108About
Dr. Barbara Snider is a distinguished Neurologist in Saint Louis, MO. Dr. Snider specializes in diagnosing, treating, and managing disorders of the brain and nervous system. With expertise in handling complex conditions like epilepsy, multiple sclerosis, and migraines, Dr. Snider employs advanced techniques and personalized treatment plans to improve patient outcomes. As a neurologist, Dr. Snider is committed to staying abreast of the latest developments in neurological research and therapies.
Education and Training
University of Texas Southwestern Medical Center Southwestern Medical School 1989
Provider Details
Expert Publications
Data provided by the National Library of Medicine- A proteasomal stress response: pre-treatment with proteasome inhibitors increases proteasome activity and reduces neuronal vulnerability to oxidative injury.
- Neurotrophin and GDNF family ligands promote survival and alter excitotoxic vulnerability of neurons derived from murine embryonic stem cells.
- Calcium in Alzheimer's disease pathogenesis: too much, too little or in the wrong place?
- Transient encephalopathy in a postoperative non-alcoholic female with Marchiafava-Bignami disease.
- Transplantation of embryonic stem cells improves nerve repair and functional recovery after severe sciatic nerve axotomy in rats.
- Cellular calcium deficiency plays a role in neuronal death caused by proteasome inhibitors.
- Translational biomarkers for accelerating drug development: From preclinical to clinical - AAPS Workshop.
- A role for the ubiquitin-proteasome system in activity-dependent presynaptic silencing.
- Optimal promoter usage for lentiviral vector-mediated transduction of cultured central nervous system cells.
- Production of lentiviral vectors for transducing cells from the central nervous system.
- Pretreatment with proteasome inhibitors protects against oxidative injuries via PPARĪ±-dependent and -independent pathways in ARPE-19 cells.
- "Will I get Alzheimer disease?" when cognitively normal patients ask to be tested for Alzheimer disease.
- Reductions of the components of the calreticulin/calnexin quality-control system by proteasome inhibitors and their relevance in a rodent model of Parkinson's disease.
- The influence of cerebrospinal fluid (CSF) biomarkers on clinical dementia evaluations.
- Proteasome inhibitors activate autophagy involving inhibition of PI3K-Akt-mTOR pathway as an anti-oxidation defense in human RPE cells.
Treatments
- Dementia
- Memory Loss
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