Dr. Joseph P. Casazza MD
Infectious Disease Specialist | Infectious Disease
1701 14th St Nw Washington DC, 20009About
Dr. Joseph Casazza is an infectious disease specialist practicing in Washington, DC. Dr. Casazza specializes in infections that are difficult to diagnose or unresponsive to treatments, such as HIV or airborne infections from a foreign country. Infectious disease specialists usually work with conditions that are not treatable by a primary physician but it is important to keep contact with the primary physician in order to receive information about the patients history and for deciding which diagnostic tests are appropriate.
Education and Training
University of Texas Southwestern Medical Center Southwestern Medical School 1995
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Infectious Disease
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Studies on the mode of sugar-phosphate product inhibition of brain hexokinase.
- Putative immunodominant human immunodeficiency virus-specific CD8(+) T-cell responses cannot be predicted by major histocompatibility complex class I haplotype.
- Decay kinetics of human immunodeficiency virus-specific CD8+ T cells in peripheral blood after initiation of highly active antiretroviral therapy.
- Monitoring HIV-specific CD8+ T cell responses by intracellular cytokine production.
- Analysis of total human immunodeficiency virus (HIV)-specific CD4(+) and CD8(+) T-cell responses: relationship to viral load in untreated HIV infection.
- A novel approach to the analysis of specificity, clonality, and frequency of HIV-specific T cell responses reveals a potential mechanism for control of viral escape.
- HIV preferentially infects HIV-specific CD4+ T cells.
- Immunologic pressure within class I-restricted cognate human immunodeficiency virus epitopes during highly active antiretroviral therapy.
- Preferential infection shortens the life span of human immunodeficiency virus-specific CD4+ T cells in vivo.
- PD-1 is a regulator of virus-specific CD8+ T cell survival in HIV infection.
- Mutation of essential catalytic residues in pig citrate synthase.
- Acquisition of direct antiviral effector functions by CMV-specific CD4+ T lymphocytes with cellular maturation.
- Immunisation with BCG and recombinant MVA85A induces long-lasting, polyfunctional Mycobacterium tuberculosis-specific CD4+ memory T lymphocyte populations.
- Early depletion of Mycobacterium tuberculosis-specific T helper 1 cell responses after HIV-1 infection.
- Differential association of programmed death-1 and CD57 with ex vivo survival of CD8+ T cells in HIV infection.
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