Dr. Raymond B Nagle MD, PHD
Pathologist | Anatomic Pathology & Clinical Pathology
1501 N Campbell Ave Tucson AZ, 85724About
Dr. Raymond Nagle is a pathologist practicing in Tucson, AZ. Dr. Nagle is a doctor who specializes in the study of bodily fluids and tissues. As a pathologist, Dr. Nagle can help your primary care doctor make a diagnosis about your medical condition. Dr. Nagle may perform a tissue biopsy to determine if a patient has cancer, practice genetic testing, and complete a number of laboratory examinations. Pathologists can also perform autopsies which can determine a persons cause of death and gain information about genetic progression of a disease.
Education and Training
Univ of Wa Sch of Med, Seattle Wa 1964
University of Washington School of Medicine 1964
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Experimental induction of rhabdomyosarcoma in mice with fractionated doses of beta-irradiation.
- EGF induces the expression of matrilysin in the human prostate adenocarcinoma cell line, LNCaP.
- N-Cadherin expression in human prostate carcinoma cell lines. An epithelial-mesenchymal transformation mediating adhesion withStromal cells.
- Promatrilysin expression is induced by fibroblast growth factors in the prostatic carcinoma cell line LNCaP but not in normal primary prostate epithelial cells.
- Altered surface expression and increased turnover of the alpha6beta4 integrin in an undifferentiated carcinoma.
- Prognostic factors in colorectal cancer. College of American Pathologists Consensus Statement 1999.
- Diagnostic frozen prostate sextant biopsies: an approach for preserving protein and RNA for additional studies.
- Aberrant expression of fibroblast growth factor receptor-1 in prostate epithelial cells allows induction of promatrilysin expression by fibroblast growth factors.
- Unique expression pattern of the alpha6beta4 integrin and laminin-5 in human prostate carcinoma.
- Investigation into the mechanism of the loss of laminin 5 (alpha3beta3gamma2) expression in prostate cancer.
- Laminin-5-mediated gene expression in human prostate carcinoma cells.
- Contractility of renal cortex following complete ureteral obstruction.
- PEAZ-1: a new human prostate neoplastic epithelial cell line.
- New molecular approaches to tissue analysis.
- Expression of the matrix metalloproteinase promatrilysin in coculture of prostate carcinoma cell lines.
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