Dr. Ronald N Cohen MD
Endocrinology-Diabetes | Endocrinology, Diabetes & Metabolism
180 Harvester Dr Suite 110 Willowbrook IL, 60527About
Dr. Ronald Cohen practices Endocrinology in Willowbrook, IL. Dr. Cohen specializes in preventing, diagnosing, and treating diseases related to hormone imbalance, and the bodys glands in the endocrine system. Endocrinologists are trained and certified to treat a variety of conditions, including menopause, diabetes, infertility, and thyroid disorders, among many others. Dr. Cohen examines patients, determines means of testing, diagnoses, and decides the best treatment methods.
Education and Training
Tel Aviv Univ- Sackler Fac Of Med- Tel Aviv- Israel 1998
Board Certification
Internal MedicineAmerican Board of Internal MedicineABIM- Endocrinology, Diabetes and Metabolism
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Partial deficiency of thyroid transcription factor 1 produces predominantly neurological defects in humans and mice.
- Determination of nuclear receptor corepressor interactions with the thyroid hormone receptor.
- A de novo mutation in an already mutant nucleotide of the thyroid hormone receptor beta gene perpetuates resistance to thyroid hormone.
- The nuclear receptor corepressors NCoR and SMRT decrease peroxisome proliferator-activated receptor gamma transcriptional activity and repress 3T3-L1 adipogenesis.
- A novel thyroid hormone receptor-beta mutation that fails to bind nuclear receptor corepressor in a patient as an apparent cause of severe, predominantly pituitary resistance to thyroid hormone.
- Nuclear receptor corepressors and PPARgamma.
- SMRT recruitment by PPARgamma is mediated by specific residues located in its carboxy-terminal interacting domain.
- SMRT recruitment by PPARgamma is mediated by specific residues located in its carboxy-terminal interacting domain.
- Altering PPARgamma ligand selectivity impairs adipogenesis by thiazolidinediones but not hormonal inducers.
- Natural angiogenesis inhibitor signals through Erk5 activation of peroxisome proliferator-activated receptor gamma (PPARgamma).
- The silencing mediator of retinoid and thyroid hormone receptors (SMRT) regulates adipose tissue accumulation and adipocyte insulin sensitivity in vivo.
- Discrepant serum and urine β-hCG results due to production of β-hCG by a cribriform-morular variant of thyroid papillary carcinoma.
- Laminin alpha4 deficient mice exhibit decreased capacity for adipose tissue
- NCoR1 and SMRT play unique roles in thyroid hormone action in vivo.
- Silencing Mediator of Retinoid and Thyroid Hormone Receptors (SMRT) regulates glucocorticoid action in adipocytes.
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