Dr. Kenneth E White M.D.
Psychiatrist | Psychiatry
24 Chestnut St Vinalhaven ME, 04863About
Dr. Kenneth White is a psychiatrist practicing in Vinalhaven, ME. Dr. White is a medical doctor specializing in the care of mental health patients. As a psychiatrist, Dr. White diagnoses and treats mental illnesses. Dr. White may treat patients through a variety of methods including medications, psychotherapy or talk therapy, psychosocial interventions and more, depending on each individual case. Different medications that a psychiatrist might prescribe include antidepressants, antipsychotic mediations, mood stabilizers, stimulants, sedatives and hypnotics. Dr. White treats conditions like depression, anxiety, OCD, eating disorders, bipolar disorders, personality disorders, insomnia, ADD and other mental illnesses.
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Expert Publications
Data provided by the National Library of Medicine- Plasma membrane Ca2+-ATPase and NCX1 Na+/Ca2+ exchanger expression in distal convoluted tubule cells.
- A novel recessive mutation in fibroblast growth factor-23 causes familial tumoral calcinosis.
- FGF23 and disorders of phosphate homeostasis.
- Genetic dissection of phosphate- and vitamin D-mediated regulation of circulating Fgf23 concentrations.
- Fibroblast growth factor-23 mutants causing familial tumoral calcinosis are differentially processed.
- Fibroblast growth factor 23 and its receptors.
- Analysis of the biochemical mechanisms for the endocrine actions of fibroblast growth factor-23.
- Extended mutational analyses of FGFR1 in osteoglophonic dysplasia.
- The role of mutant UDP-N-acetyl-alpha-D-galactosamine-polypeptide N-acetylgalactosaminyltransferase 3 in regulating serum intact fibroblast growth factor 23 and matrix extracellular phosphoglycoprotein in heritable tumoral calcinosis.
- Loss of DMP1 causes rickets and osteomalacia and identifies a role for osteocytes in mineral metabolism.
- Molecular insights into the klotho-dependent, endocrine mode of action of fibroblast growth factor 19 subfamily members.
- Two novel GALNT3 mutations in familial tumoral calcinosis.
- Molecular genetic and biochemical analyses of FGF23 mutations in familial tumoral calcinosis.
- A rat model of chronic kidney disease-mineral bone disorder.
- Molecular analysis of DMP1 mutants causing autosomal recessive hypophosphatemic rickets.
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