Dr. Arnaldo A Arbini MD
Hematologist-Pathologist | Hematology
560 1st Ave Th 381 New York NY, 10016About
Dr. Arnaldo Arbini is a hematology pathologist practicing in New York, NY. Dr. Arbini specializes in reviewing blood samples and interpreting laboratory results. These results give them an idea of whether certain diseases are malignant or not, in order for them to properly determine what exact treatment is needed. Hematology pathologists are normally experts in leukemia, lymphoma, anemia, hemophilia and many other blood diseases.
Education and Training
Pontificia Univ Javeriana, Fac De Med, Bogota, Colombia 1984
Board Certification
PathologyAmerican Board of PathologyABP
Provider Details
Expert Publications
Data provided by the National Library of Medicine- Optimal dosing and triggers for prophylactic use of platelet transfusions.
- Angioleiomyoma of the patellar tendon sheath. Case report.
- Down-regulation of BRCA2 expression by collagen type I promotes prostate cancer cell proliferation.
- Up-regulation of Skp2 after prostate cancer cell adhesion to basement membranes results in BRCA2 degradation and cell proliferation.
- Constitutive activation of MAPK/ERK inhibits prostate cancer cell proliferation through upregulation of BRCA2.
- Mitochondrial DNA depletion reduces PARP-1 levels and promotes progression of the neoplastic phenotype in prostate carcinoma.
- Mitochondrial DNA depletion in prostate epithelial cells promotes anoikis resistance and invasion through activation of PI3K/Akt2.
- Stress-related mitochondrial components and mitochondrial genome as targets of anticancer therapy.
- Bone marrow uptake of ferumoxytol: a preliminary study in healthy human subjects.
- Mitochondrial DNA depletion sensitizes cancer cells to PARP inhibitors by translational and post-translational repression of BRCA2.
- Rapid and robust reversion to essential thrombocythemia on treatment with Decitabine in a case of hydroxyurea-induced t-MDS/AML.
- [Corrigendum] Constitutive activation of MAPK/ERK inhibits prostate cancer cell proliferation through upregulation of BRCA2.
- Mitochondria and cancer chemoresistance.
- Determinants of plasma factor VIIa levels in humans.
- Molecular analysis of Polish patients with factor VII deficiency.
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